Titlebook: Detection of Mitochondrial Diseases; Frank Norbert Gellerich,Stephan Zierz Book 1997 Springer Science+Business Media Dordrecht 1997 genome

Infinitesimal

Das Ende der Strukturkategorie Geschlecht?s obtained by comparing the ADP supply from mitochondrial kinases to oxidative phosphorylation with that of non-bound, extramitochondrially located kinases. This approach allowed us to estimate the ADP diffusion gradients which were present across the outer membrane and between the compartment forme

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Women’s Political Representation in Brazilurine, carnitine, phosphocreatine, N-acetylcysteine, Trolox-C and ruthenium red, implying a role for reactive oxygen species and calcium-ions in the in vitro damage at low temperature to oxidative phosphorylation..In heart mitochondria oxphos with pyruvate and palmitoylcarnitine was most labile foll

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José Álvaro Moisés,Beatriz Rodrigues Sanchezized fiber could immediately be detected during the course of measurements..In 4 patients with mitochondrial encephalomyopathy (MEM) the respiration pattern indicated a specific mitochondrial enzyme defect, which was confirmed in every patient by measurements of the individual enzymes (one patient w

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https://doi.org/10.1007/978-3-030-53130-0om ischemic hearts or from hearts preserved by immersion showed a decrease of the apparent K. for ADP, and a complete loss of the stimulatory effect of creatine. In these fibers, we could observe that the outer mitochondrial membrane was damaged. In conclusion, the results of this study show that as

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Diagnosis of defects in oxidative muscle metabolism by non-invasive tissue oximetrymonstrates specific abnormalities in a variety of mitochondrial disorders, indicating abnormal oxygen utilization, and will be a useful addition to the clinical investigation of such disorders. (Mol Cell Biochem . 7–10, 1997)