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Titlebook: Cytokine Yearbook Volume 1; An Official Publicat S. Pestka,H. Schellekens,P. Wussow Book 1996 Kluwer Academic Publishers 1996 cytokine.cyto

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书目名称Cytokine Yearbook Volume 1
副标题An Official Publicat
编辑S. Pestka,H. Schellekens,P. Wussow
视频video
图书封面Titlebook: Cytokine Yearbook Volume 1; An Official Publicat S. Pestka,H. Schellekens,P. Wussow Book 1996 Kluwer Academic Publishers 1996 cytokine.cyto
描述The justification for yearbooks is greater than ever as weapproach the third millennium, overwhelmed with information. Thisfirst edition of the .Cytokine Yearbook. summarizes the latestadvances in the revolutionary field of cytokine research. The work isnot a comprehensive reference work, but covers a selection of currentthemes. The intention is to keep paying attention to current topics inthe .Yearbooks. to come. .The editors invited a number of distinguished colleagues, who areinternational experts in their specific fields, resulting in a highscientific level of the contributions. .This .Yearbook. is required reading for every scientist andphysician working in the field of cytokines.
出版日期Book 1996
关键词cytokine; cytokines; diseases; infectious disease; interferon
版次1
doihttps://doi.org/10.1007/978-94-009-1616-6
isbn_softcover978-94-010-7221-2
isbn_ebook978-94-009-1616-6
copyrightKluwer Academic Publishers 1996
The information of publication is updating

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International and Cultural Psychologyof research that are beginning to be elucidated. Signaling molecules other than the JAKs and STATs have also been found to be activated following interferon binding. In addition, the induction of type I interferon stimulated genes by double-stranded RNA in the absence of interferon provides another
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https://doi.org/10.1007/978-3-322-95902-7f the IFN-. receptor (IFN-.sR). These agents ameliorated disease development in a variety of experimental autoimmune diseases in rodents. In a mouse model for the human disease Myasthenia gravis, IFN-. was found to reduce both the incidence and progression of the disease. Treatment of R. aurantiacus
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https://doi.org/10.1007/978-1-4614-6303-0 Most of you knew him very well because of his work in the interferon field. He regularly attended our interferon meetings since the very beginning. However, until recently, only a few close friends knew about his serious illness as Giovanni never exhibited his illness. He managed not only to carry
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International and Cultural Psychologyg of interferon to a transmembrane receptor stimulates the activation of associated tyrosine kinases of the Janus kinase (JAK) family. Subsequently, latent cytoplasmic transcription factors are activated by tyrosine phosphorylation and function as signal transducers and activators of transcription (
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International and Cultural Psychologyractory state in the reponse to interferon (IFNs) and antibodies in treated patients; Workshop 7, IFNs, multiple sclerosis, and the nervous system; Workshop 9, Viral inhibition of the response to IFNs and other cytokines; Workshop 10, Cell growth inhibition by IFNs and other cytokines; Workshop 12,
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