Titlebook: Cyclic Nucleotides; Part II: Physiology John W. Kebabian,James A. Nathanson Book 1982 Springer-Verlag Berlin Heidelberg 1982 biochemistry.

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Lecture Notes in Networks and Systems gastric glands. Studies on isolated parietal cells emphasize the interaction of cell function with cyclic nucleotide phosphodiesterase inhibition and adenylyl cyclase regulation. The authors conclude that the evidence supports the concept of separate parietal cell receptors for the acid secretagogu

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0171-2004 renocep­ tor, but he did not provide any insight into how stimulation of the receptor produces tissue-specific physiological responses. The discovery of cyclic 978-3-642-68395-4978-3-642-68393-0Series ISSN 0171-2004 Series E-ISSN 1865-0325

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Regulation of Lipid Metabolism by Cyclic Nucleotidesbound adenosine which exerts an inhibitory constraint on basal adenylate cyclase activity. Thyroid hormones regulate adenylate cyclase activity of adipocytes by affecting the coupling of the hormone-receptor complexes to adenylate cyclase. Growth hormone also activates adenylate cyclase through a pr

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Regulation of the Cell Cycle and Cellular Proliferation by Cyclic Nucleotidesell proliferation. Most attention will focus upon the studies that have appeared during the past four years since there have been a number of critical reviews covering prior studies (Abell and Monahan 1973; Ryan and Heidrick 1974; Pastan and Johnson 1974; Pastan et al. 1975; Chlapowski et al. 1975;