Titlebook: Current Topics in Complement II; John D. Lambris Book 2008 The Editor(s) (if applicable) and The Author(s), under exclusive license to Spr

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Chuanqi Wang,David Arellano,Roger Meier upstream components of the innate immune system, recognizing exogenous structures as well as endogenous ligands. They act partly independent in the inflammatory network, but also have several cross-talk mechanisms which are under current investigation. Complement is an essential part of innate immu

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Dedication

Food Intake Regulation by Central Complement System,e response. C3a, C5a and their receptors have been revealed to be present in the central nervous system (CNS) as well as the peripheral immune system. We found that centrally administered C3a suppresses food intake, while C5a stimulates food intake, and their food intake regulation may be associated

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The Role of Membrane Complement Regulatory Proteins in Cancer Immunotherapy,se. Complement activation mediated by anti-tumor mAbs can result in direct tumor lysis or enhancement of antibody-dependent cellular cytotoxicy. Chemotaxis of phagocytic cells by complement activation products C5a is also required for certain cancer immunotherapy such as combined β -glucan with anti

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Subversion of Innate Immunity by Periodontopathic Bacteria via Exploitation of Complement Receptor-ng disease. Here we review subversive interactions of Porphyromonas gingivalis, a major periodontal pathogen, with the complement receptor-3 (CR3; CD11b/CD18) in monocytes/macrophages. Through its cell surface fimbriae, P. gingivalis stimulates Toll-like receptor-2 (TLR2) inside-out signaling which

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