Titlebook: Current Perspectives in Hepatology; Festschrift for Hyma Leonard B. Seeff (Chief, Professor of Medicine),Ja Book 1989 Springer Science+Busi

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Autoimmune Chronic Active Hepatitis picture (Table I). The clinical presentation, natural history, and response to treatment vary widely among the different etiologies. Clinically, the syndrome is defined by persistent necroinflammation in the liver for more than 6 months, as reflected by persistent elevation of serum aminotransferas

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Chronic Lupoid Hepatitis: Autoimmune Chronic Active Hepatitismilar findings a year later. Similarly, 1 year after Joske and King. reported the lupus erythematosus (LE) cell phenomenon in active chronic viral hepatitis, Zimmerman and colleagues. confirmed that the LE cell phenomenon was a feature in some forms of chronic hepatic disease.

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Primary Biliary Cirrhosis: Clinical Aspects ducts, portal inflammation and scarring, and the eventual development of cirrhosis and liver failure. The diagnosis is usually made in a middle-aged woman (although the age range is 20–80 years) who initially has fatigue or itching or who is asymptomatic and has been found to have unexplained hepat

STAT

Primary Biliary Cirrhosis: Pathologylesion and results in a progressive disappearing bile duct syndrome. Although there is clinical and/or biochemical evidence of a cholestatic syndrome from the time of diagnosis, morphologic evidence of cholestasis does not appear until there has been extensive loss of intrahepatic bile ducts; jaundi

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GLEAN

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Umfassende Theorien der Sozialwissenschaften progression was just as likely following asymptomatic as following overt icteric hepatitis, it became apparent that such mild cases could not be ignored.. Skepticism then shifted to another level; i. e., even if NANBH frequently results in chronic hepatitis, as judged by alanine aminotransferase (A

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