Titlebook: Corynebacterium diphtheriae and Related Toxigenic Species; Genomics, Pathogenic Andreas Burkovski Book 2014 Springer Science+Business Media

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Siwei Qiang,Yongkun Wang,Yaohui Jintic C-domain) that catalyses the transfer of an ADP-ribosyl group of NAD. to a post-translationally modified histidine (diphthamide) residue on eukaryotic elongation factor 2 (eEF2) and inhibits protein synthesis. Fragment B (FB) consist of the carboxy terminal receptor-binding R-domain, and the tra

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macabre

Ningning Ma,Hai-Tao Zheng,Xi Xiaohumans, e.g. in case of diphtheria. However, besides diphtheria toxin action, information about molecular mechanisms of host pathogen interaction is rare. The recent availability of genome sequence information gave new impetus to the characterization of putative virulence factors of ., . and . and t

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Dawei Gao,Yongxin Tong,Yudian Ji,Ke Xuential to re-emerge to epidemic proportions in areas where it was previously thought to be under control. Ongoing monitoring and surveillance is therefore essential and in general follows the principles of surveillance utilised for most other vaccine preventable infections, but with some specific ad

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Keyphrase Extraction Using Knowledge Graphsents methods for antimicrobial susceptibility testing of . and recommendations with the focus on discrepancies concerning testing conditions and interpretive breakpoints. Resistance of . isolated in different regions of the world has been described including multidrug-resistant strains.

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Christopher D. Hollings,Mark V. Lawsonemical and genetic tools. Multiple compatible plasmid origins are now available and the methods used to introduce DNA to . have been both improved and expanded to include conjugation. In the last decade, a technique to perform transposon mutagenesis in . was developed as well as phage-based vectors