Titlebook: Core Concepts in Acute Kidney Injury; Sushrut S. Waikar,Patrick T. Murray,Ajay K. Singh Book 2018 Springer Science+Business Media, LLC, pa

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Tubular Physiology in Acute Kidney Injury: Cell Signalling, Injury and Inflammationlease from growth-arrested, injured or necrotic cells. The roles of leukocyte subsets, activation of toll-like receptors and complement activation in mediating inflammatory activation, tissue damage and eventual repair in the injured kidney will also be explored.

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Acute Kidney Injury and Cancer: Incidence, Pathophysiology, Prevention/Treatment, and Outcomess. Identification of at-risk patients allows measures to reduce the risk of AKI. Where AKI does occur, early diagnosis and treatment is important—this is most likely to happen when there is close collaboration between oncologists and nephrologists.

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Definition and Classification of Acute Kidney Injury.5 mL/kg per hour for more than 6 h. Progressive stages of AKI severity are defined by larger increases in serum creatinine, longer durations of oliguria or anuria, or need for renal replacement therapy. Although these definitions are useful in epidemiologic studies and clinical trials, their utilit

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Diagnostic Approach: Differential Diagnosis, Physical Exam, Lab Tests, Imaging, and Novel Biomarkersg “subclinical AKI.”.The approach to any patient with AKI requires detailed attention to physical exam findings (which may often be subtle), several blood and urine tests (including examination of the urinary sediment), and potentially a variety of imaging test. Urinary indices such as FENa and FEUr

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Contrast-Associated Acute Kidney Injury concern by providers for the development of CA-AKI. CA-AKI is one of the few potentially preventable forms of renal injury; as a result, there has been substantial interest in identifying strategies to reduce the risk of this condition. Past research of multiple pharmacological and non-pharmacologi

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Acute Kidney Injury and Liver Disease: Incidence, Pathophysiology, Prevention/Treatment, and Outcome Many of the traditional means of differential diagnosis, including measuring the fractional excretion of sodium and examining urine sediment, perform suboptimally in cirrhosis. Recent studies have identified multiple urinary biomarkers associated with structural kidney injury that have shown promis

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Drug-Induced Acute Kidney Injury therapeutic agents, reducing exposure in at-risk patients. Once identified, the medication or agent can be withheld or dose-reduced, thereby significantly impacting and potentially reducing the duration and severity of kidney injury.

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Sepsis and Acute Kidney Injury: Epidemiology, Pathophysiology, Diagnosis, and Managementent options to target SA-AKI exist yet, the general principles in the evaluation and management of SA-AKI are aimed at early fluid resuscitation and antibiotic administration, followed by a strategy of limiting injury, avoiding life-threatening complications, eliminating any potential contributors t

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