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Titlebook: Congestive Heart Failure; Proceedings of the S Joel Morganroth (Prof. of Med. and Pharm.),E. Neil Conference proceedings 1987 Martinus Nijh

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Roberto I. Troisi,Vincenzo Scuderir drugs that may have a direct action on cardiac electrophysiologic or hemodynamic parameters. In fact, all antiarrhythmic agents, though prescribed to suppress cardiac arrhythmias, have been documented to have the potential to aggravate or provoke arrhythmias..
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Kathryn J. Wood,Masaaki Zaitsu,Ryoichi Goto obtained from the National Center for Health Statistics indicate that the number of deaths due to congestive heart failure (CHF) has increased more than four-fold; for example, the number of deaths in which CHF was considered to be the underlying cause increased from about 6000 in 1955 to over 30,0
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Nicole M. Valenzuela,Elaine F. Reedhe least of which are an adequate understanding of the etiology of the underlying process of myocardial failure, a dissociation of various endpoints from one another, such as symptomatology and mortality and an inability to define the factors which lead to progression of the primary disease.
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Roberto I. Troisi,Vincenzo Scuderifor an appropriate animal model for the disease. In the various clinical conditions called heart failure, reflex mechanisms are triggered by the body in an attempt to maintain hemeostasis in the face of a failing circulation. This further complicates the picture as sympathetic, renin-angiotensin, an
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John D. Terrace,Gabriel C. Onisculier recognition of the provoking pathology and improved hospital and intensive care treatment. In spite of multiple interventional studies directed to the correction of hemodynamic abnormalities and the improvement of functional capacity, patients with heart failure continue to have a high annual m
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