Titlebook: Clues in the Diagnosis of Non-tumoral Testicular Pathology; Manuel Nistal,Pilar González-Peramato,Álvaro Serra Book 2017 Springer Internat

TOXIC

Differential Diagnosis of Tumors in the Adrenogenital Syndrome,nhibin, synaptophysin, and CD56) although there are differences in the intensity of positivity and the distribution of the expression – focal versus diffuse in some cases. The absence of androgen receptors and expression of INSL3 constitute the most relevant histological differential fact.

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Differential Diagnosis of Sertoli Cell Nodules,ord tumor with annular tubules, Sertoli cell tumor “not otherwise specified,” and others like gonadoblastoid testicular dysplasia, Sertoli-Leydig hamartoma, and Sertoli cell adenoma. Differential diagnosis with Sertoli cell tumors is especially important as these often are associated with syndromes

FAR

Meaning of the Finding of Testicular and Paratesticular Calcifications,he testicular parenchyma. Two types, classic (more than five microliths by ultrasound field) and limited (five or less microliths per ultrasound field) are distinguished. The macrocalcification is defined as any focus of “coarse” calcification that is larger than 1 mm and separated from any intrates

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The Most Frequent Histological Findings in the Adult Testis When Testicular Descent Was Performed ided testes are the result of hypogonadism from fetal life, as can be seen in the little proliferation of germ cells and also in postnatal life coinciding with the mini-puberty due to incomplete transformation of gonocytes in Ad spermatogonia. The Ad spermatogonia are the stem cells, and a low number

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Testicular Dysgenesis Syndrome (TDS),es the testicular histological findings (mixed atrophy, hypoplastic tubules, Sertoli cell nodules, granular changes in Sertoli cells, tubules with germ cell hypoplasia, microlithiasis, focal Leydig cell hyperplasia, and germ cell neoplasia in situ) that enable to suggest that a patient has a testicu

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Value of Testicular Biopsy in Nonobstructive Azoospermia, the classification of these lesions, the starting point to know whether other genes are involved in spermatogenesis, and the evaluation of noninvasive techniques such as detection of germ cell-specific mRNA traces in seminal plasma to predict the state of spermatogenesis. The most common histologic

Arteriography

Obstructive Mechanism Lesions Simulating Primary Testicular Lesions,uction can be distinguished: young spermatid sloughing, late primary spermatocyte sloughing, early primary spermatocyte sloughing, and hypospermatogenesis associated with primary spermatocyte sloughing. In addition to these primary testicular lesions, those other lesions that most often lend to a di