Titlebook: Clostridial Neurotoxins; The Molecular Pathog Cesare Montecucco Book 1995 The Editor(s) (if applicable) and The Author(s), under exclusive

legacy

Botulism: The Present Status of the Disease,otulism is the result of ingestion of food containing neurotoxin produced by .. The disease may also result from toxin produced in vivo by organisms growing in an infected wound (wound botulism) or colonizing the intestinal tract (infant botulism).

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Cellular Immunology of Tetanus Toxoid,immunogens. This review deals with two related aspects of TeNT as an antigen: (a) immunological responses at the T and B cell level to tetanus in humans and the possibility of exploiting tetanus in the wider context of immunotherapy, and (b) some aspects of the cell biology and biochemistry of TeNT processing in human B lymphoblastoid cells.

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forebear

Schizophrenia Treatment Outcomestion with consumption of sausages, and thus the name botulism (derived from ., Latin for sausage) was given to the disease (. and . 1988). Foodborne botulism is the result of ingestion of food containing neurotoxin produced by .. The disease may also result from toxin produced in vivo by organisms g

Galactogogue

https://doi.org/10.1007/978-3-319-73993-9effective immunogen, and how we might be able to exploit the biochemistry and physiology of antigen-presenting cells (APC) to make yet more effective immunogens. This review deals with two related aspects of TeNT as an antigen: (a) immunological responses at the T and B cell level to tetanus in huma

infarct

https://doi.org/10.1007/978-3-319-73993-9 command attention and concern by those around the victim. In earlier times help might have been sought from the shaman or witch doctor, whose spells or potions provided little comfort from the symptoms and eventual death. As modern medicine developed in the last two centuries even well-trained phys

Tartar

Why Are Delusions Pathological?nineteenth century, when . (1897) related botulism to a toxin produced by ., an anaerobic bacterium. The analysis of botulism outbreaks has led to the recognition that human intoxication may be caused by BoNT types A, B, E, and F (. et al. 1983; . 1983). There are three other antigenically distinct

土产

https://doi.org/10.1007/978-3-319-73993-9nguished on the basis of their reactions with specific antibodies and are the causative agents of the disease botulism. These toxins act on the peripheral nervous system where they block neuromuscular transmission and induce widespread flaccid paralysis (. 1986; . 1991). Tetanus toxin, on the other