Titlebook: Clinician‘s Guide to Mycosis Fungoides; Pooya Khan Mohammad Beigi Book 2017 Springer International Publishing AG 2017 mycosis fungoides.sk

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https://doi.org/10.1007/978-3-319-41024-1Staging of mycosis fungoides (MF) can be done through the TNMB (tumor-node-metastasis-blood) classification system, which stages the disease based on the extent of skin, lymph node, viscera, and blood involvement. Of note, stages IA to IIA are also referred to as early MF. Advanced stage is IIB to IV.

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https://doi.org/10.1007/978-1-940033-60-0This chapter presents a review of the literature of treatment outcomes in various studies of patients with Mycosis Fungoides and Sézary syndrome at different disease phases. Treatments included psoralen and ultraviolet A (PUVA), narrow band ultraviolet B (NB-UVB), and interferon (IFN).

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Simon Badger,Johannes Henn,Simone ZoiaThe following case reports are detailed records of some patients that were also part of the author’s previously unpublished study. This section aims to take a closer look at some of the individual cases including patient background and history, signs and symptoms, laboratory and medical examinations, as well as diagnosis and treatment.

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StagingStaging of mycosis fungoides (MF) can be done through the TNMB (tumor-node-metastasis-blood) classification system, which stages the disease based on the extent of skin, lymph node, viscera, and blood involvement. Of note, stages IA to IIA are also referred to as early MF. Advanced stage is IIB to IV.

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青少年

Clinical Research Case DescriptionsThe following case reports are detailed records of some patients that were also part of the author’s previously unpublished study. This section aims to take a closer look at some of the individual cases including patient background and history, signs and symptoms, laboratory and medical examinations, as well as diagnosis and treatment.

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Etiologyon its possible genetic, environmental, and immunologic etiologies. Current hypotheses of MF include genetic and cellular signaling abnormalities such as clonal abnormalities and changes in the expression of adhesion molecules and cytokines. Alterations in the immunological milieu have also been implicated in the pathogenesis of MF.

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