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Titlebook: Clinical Rheumatology; Rohini Handa Book 2021 The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer Nature

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,Tödliche Vergiftung durch Nitrose Gase,from subacute and chronic LBP. Overreliance on investigations and X-rays should be avoided. Bed rest should be kept to a minimum. The evidence base supporting many interventions like firm mattresses, lumbar belts, traction, epidural injections, etc. is weak and contradictory. NSAIDs are employed for
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Barbiturat-(Soneryl-)Vergiftung,ns. OA should not be dismissed as mere aging. It is a disease for which several treatment options exist. Symptoms and radiologic changes often do not correlate in OA. Exercise plays a vital role in the management of OA. Nutraceuticals like glucosamine, chondroitin, curcumin derivatives, etc. have vi
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,Tödliche Vergiftung durch Nitrose Gase,andates search for comorbidities like metabolic syndrome, hyperlipidemia, diabetes, etc. Gout typically causes mono−/oligoarticular disease—red, hot joint. The differential diagnosis of a red, hot joint includes gout and septic arthritis. Gout is exceedingly rare in premenopausal women. Ordering SUA
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,Tödliche Vergiftung mit Pantocain,ile (aseptic), asymmetric oligoarthritis, predominantly affecting the lower limb joints. ReA may be self-limited or pursue a relapsing course. Nonsteroidal anti-inflammatory drugs are used as the first-line treatment with disease-modifying antirheumatic drugs reserved for those with persistent protr
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,Tödliche Vergiftung mit Pantocain,udicious investigations. The clinical picture may evolve over time. The patient’s history and examination should be revisited at regular intervals. Clinical correlation is a must. Caution should be exercised while interpreting some serological tests because of false-positive and false-negative resul
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Barbiturat-(Soneryl-) Vergiftung,arthritis of unknown cause where the age of onset is before the 16th birthday. The disease duration should be more than 6 weeks. The clinical manifestations, assessment, and treatment of JIA are different from adult-onset arthritis. Active disease persisting into adulthood may be seen in more than o
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