Titlebook: Chronic Kidney Disease and Hypertension; Matthew R. Weir,Edgar V. Lerma Book 2015 Springer Science+Business Media New York 2015 BP Monitor

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Reflections on a Troubled World Economyhe availability of proven therapeutic approaches including lifestyle modification and the use of antihypertensive drugs, control rates for hypertension remain unsatisfactory across the globe. While the majority of patients can be controlled with adequate combination therapy of drugs with different m

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Key Currencies and Financial Centresll or is reduced insufficiently during treatment. Treatment resistance, therefore, is one of the challenges of contemporary hypertension research. Since the autonomic system plays a major role in the pathogenesis of hypertension, in particular when this is resistant to drug treatment, investigators

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Key Currencies and Financial Centresoviders. The idea of a vaccine for hypertension that could ameliorate or cure the disease with a single procedure is innately appealing. The discovery that renin increased blood pressure raised the possibility that renin could be the key to the etiology of hypertension. Administering renin from one

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https://doi.org/10.1007/978-1-349-06892-0, white-coat hypertension, while also being well described, is generally accepted as exhibiting greater risk than normotension. More recently, the identification of masked hypertension (reverse white-coat hypertension using older terminology) has added to the complexity of the hypertensive phenotype

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analogous

No More Business as Usual: Viet Nam, The etiology of primary (essential) hypertension is complex, and consists of genetic, maternal–fetal, and environmental factors. Ideally, one would like to identify modifiable risk factors that could either prevent or treat hypertension. An elevated serum uric acid, which is a clearly amendable ris

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You Must Be Mad: UN Headquarters,the pathogenesis of preeclampsia (PE) have yet to be fully elucidated. PE affects the vasculature of many target maternal organs including the kidney, liver, and brain. Growing evidence supports the concept that the placenta plays a central role in the pathogenesis of PE and that reduced uteroplacen

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APEX

https://doi.org/10.1007/978-3-030-77063-1ed a significant heritable component to the most common form, essential hypertension. Several strategies have been developed to identify a genetic variation that is associated with hypertension. Genome-wide association studies (GWAS) were developed to identify particular genetic markers (single-nucl

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,Surviving in the People’s Paradise: DPRK,tors. These conditions include primary aldosteronism and some forms of congenital adrenal hyperplasia, Cushing’s, and pheochromocytoma/paraganglioma syndromes, respectively. One needs to remember that these conditions are rare, but associate with significant morbidity and need to be included in the