Titlebook: Chromosome 12 Aberrations in Human Solid Tumors; Cytogenetics and Mol J. Bullerdiek,S. Bartnitzke Conference proceedings 1994 Springer-Verl

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Frankophone Literatur aus dem Maghreb,he breakpoints and the chromosomal localization of oncogenes or suppressor genes has been of particular interest. In this region, four oncogenes (.) and two putative suppressor genes (., CHOP) have been localized (Fig. 1). The main results obtained concerning the function of these genes during norma

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https://doi.org/10.1007/978-3-322-97042-8rcomas stop proliferation after 3–4 in vitro passages, not allowing sufficient material for molecular analyses by in vitro preparation to be obtained (for PSA, see Stern et al. 1990), One alternative to overcome the problem of replicative senescence (Hayflick and Moorhead 1961) of tumor cells in cul

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Redeploying Urban Infrastructuretics to the more detailed description and understanding of oncogenesis (Sandberg 1990; Mitelman 1991; Solomon et al. 1991). Although fundamental genetic analysis of leukaemias and lymphomas has advanced more rapidly in the past than that of solid tumours, extensive knowledge about solid tumours has

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Shifting the State from Left to Right,liposarcoma, for instance, the primary cytogenetic change is mostly the highly consistent reciprocal translocation t(12;16)(q13;p11) or the more complex three-way translocation also involving chromosome 1 (Turc-Carel et al. 1986a; Ohjimi et al. 1992). Cytogenetic analysis of lipomas, a subgroup of b

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