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Titlebook: Cholesterol; Interactions with Te Jens Møller Book 1987Latest edition Springer-Verlag Berlin Heidelberg 1987 biochemistry.cardiovascular.he

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Comments on Photographs,I will now briefly summarize the underlying biochemical processes that lead to a deterioration of circulation and to its restoration by testosterone.
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https://doi.org/10.1007/978-3-642-95409-2nsulin-dependent, juvenile-onset diabetes can oxidize ketone bodies, built up from acetic acid units. This is an attempt to provide an alternative, noncarbohydrate energy source. If ketone-body production exceeds the rate at which dissimilation can be carried out by the tissues, then ketone bodies accumulate in the blood (ketosis).
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https://doi.org/10.1007/978-3-642-71600-3biochemistry; cardiovascular; heart disease; physiology; vascular disease
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978-3-642-71602-7Springer-Verlag Berlin Heidelberg 1987
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Fat Burns in the Flame of Carbohydrate, and create conditions in which there is insufficient oxaloacetic acid for the oxidation of acetyl CoA, resulting in an increased plasma lipid concentration and increased cholesterol level. This is in full agreement with established biochemical principles, as shown by the following quotations: “.. T
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Free Fatty Acid Metabolism,one (see, for instance, Breier et al. 1985). The intracellular hormone-sensitive lipase of adipose tissue catalyses the breakdown of stored triglycerides into glycerol and fatty acid with the latter entering the circulation. The hormone-sensitive lipase is activated by catecholamines and Cortisol. (
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The Nature of Cardiovascular Disease,s in the same sense as, for example, infections, which have a clear bacteriological etiology and can thus be treated and cured with antibiotics. .. It lies in the predominance of the influence of catabolism, represented by catecholamines and cortisol, over the role of anabolism, represented by testo
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