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Titlebook: Cholesterol; Interactions with Te Jens Møller Book 1987Latest edition Springer-Verlag Berlin Heidelberg 1987 biochemistry.cardiovascular.he

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Uwe M. Borghoff,Johann H. Schlichterel of cholesterol. Testosterone, on the other hand, activates lipoprotein lipase, leading the stream of triglycerides in the opposite direction, thereby reestablishing normal fatty acid metabolism, normal carbohydrate metabolism, and normal levels of cholesterol.
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K. Peikenkamp,J. Natrup,K. Nicolreatment is started, but are willing to run the risk of developing the side effects, since in the hospital they have been threatened with amputation. By no means all female patients experience side effects, which may be avoided by decreasing the dosage when improvement of circulation begins.
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Treatment of CVD with Testosterone,reatment is started, but are willing to run the risk of developing the side effects, since in the hospital they have been threatened with amputation. By no means all female patients experience side effects, which may be avoided by decreasing the dosage when improvement of circulation begins.
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Book 1987Latest edition therapeutically by several clinicians, especially in Germany. However, with the outbreak of the Second World War, international scientific research was effectively brought to an end. Using testosterone extracted from postmortem material, workers at the clinic of the famous Danish surgeon Thorkild R
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Diabetic States and CVD,nsulin-dependent, juvenile-onset diabetes can oxidize ketone bodies, built up from acetic acid units. This is an attempt to provide an alternative, noncarbohydrate energy source. If ketone-body production exceeds the rate at which dissimilation can be carried out by the tissues, then ketone bodies accumulate in the blood (ketosis).
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Einleitung und Aufgabenstellung, This dispute, which has often taken on a bitter tone, still continues and has so far failed to produce any results beneficial to patients. In my opinion high cholesterol concentration is more a . of a deterioration in circulation than a factor causing CVD. This is certainly the case where raised ch
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Uwe M. Borghoff,Johann H. Schlichter and create conditions in which there is insufficient oxaloacetic acid for the oxidation of acetyl CoA, resulting in an increased plasma lipid concentration and increased cholesterol level. This is in full agreement with established biochemical principles, as shown by the following quotations: “.. T
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