Titlebook: Cholecystokinin Antagonists in Gastroenterology; Basic and Clinical S Guido Adler,Christoph Beglinger Book 1991 Springer-Verlag Berlin Heid

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Inhalte festhalten und verwaltencids into the duodenum. Together with monoglycerides and phospholipids, bile acids form mixed micelles to facilitate fat absorption. Eventually, 95% of them are reabsorbed by the terminal ileum to finish the enterohepatic cycle [4]. Since bile acids presumably get in contact with intestinal CCK cell

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Beispiel: Recherche in der Praxisch these actions may be mediated have been identified in multiple brain regions and at various sites in the gastrointestinal tract. This chapter focuses on the anatomy, pharmacological characterization, and functional significance of a population of CCK receptors localized to the circular muscle lay

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Book 1991p-to-date information in cholecystokinin (CCK) research, especially focusing on the development and characterization of CCK antagonists. The book contains chapters on the synthesis, biochemical and pharmacological characterization of potent and selective CCK antagonists as well as physiological appl

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Biological Actions of CCK in the Central Nervous Systemnant molecular form of CCK in the brain is CCK. (CCK., the eight amino acids at the C terminus of CCK), which contains a sulfated tyrosine residue [14,54]. However, the unsulfated form (CCK.) has been detected, in addition to CCK. [54]. Overall, CCK. is only present in the brain in small (2%-5% of CCK immunoreactivity) amounts [54].

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Boc-Tyr(SO3H)-Nle-Gly-Trp-Nle-Asp-2-Phenylethyl Ester — JMV180: A Unique CCK Analogue with Different-Asp-NH.], led to CCK antagonists both in the peripheral system [1] and in the CNS [2,3]. We report here on the modulations in activity which resulted from suppression of the C-terminal primary amide function of CCK, and on its pharmacological consequences.

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