Titlebook: Chemotactic Cytokines; Biology of the Infla J. Westwick,I. J. D. Lindley,S. L. Kunkel Book 1991 The Editor(s) (if applicable) and The Autho

Metastasis

Lecture Notes in Physics Monographsin an HL60 cDNA library screened with oligonucleotide probes designed from the N-terminal amino acid sequence. Sequencing of the cDNA reveals a protein of 225 amino acids with significant nucleotide homology to cathepsin G and human neutrophil elastase.

Anthem

木讷

Human Neutrophil Granule Cationic Protein CAP37 is a Specific Macrophage Chemotaxin that Shares Homin an HL60 cDNA library screened with oligonucleotide probes designed from the N-terminal amino acid sequence. Sequencing of the cDNA reveals a protein of 225 amino acids with significant nucleotide homology to cathepsin G and human neutrophil elastase.

Bone-Scan

NAP-1/IL-8 in Rheumatoid Arthritis, inflammatory mediator. Further actions which could be of great importance in inflammatory processes are chemotactic attraction of basophils, the triggering of IL-3 primed basophils to release leukotrienes and histamine (10), spasmogenic activity on airway smooth muscle (11), and effects on PMN adherence to endothelial cells (12).

Frequency-Range

MULTI

Consensus

Book 1991a peptide consisting of 72 amino acids which was a potent activator of neutrophils and a chemotactic agent for lymphocytes. The first symposium of this series was held at the Royal College of Surgeons of England in December 1988, entitled Novel Neutrophil Stimulating Peptides, and brought together t

Instrumental

,Interleukin-8 —A Mediator of Inflammatory Lung Disease?,ures of asthmatic airways. It is known that intensification of BHR follows antigen challenge in atopic asthmatics (4) and it has been widely presumed that inflammatory events during late-onset reactions may determine the changes in airway reactivity (5).

foreign

Some Aspects of NAP-1 Pathophysiology: Lung Damage Caused by a Blood-Borne Cytokine,our ., one can suggest that NAP-1 and also the recently discovered NAP-2 (4) and NAP-3 (5) (all three belong to the C-X-C cytokine family, exhibit considerable structural homology and have analogous activities .) could be responsible for the induction of the inflammatory cellular infiltrates dominated by neutrophils.

ovation

https://doi.org/10.1007/978-3-540-47295-7 evolves. This characteristic of an inflammatory lesion is a good example of the host’s ability to synthesize specific agents, which can elicit specific white blood cells, at specific time intervals of an inflammatory response.