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Titlebook: Cerebrovascular Transport Mechanisms; International Congre Konstantin-Alexander Hossmann,Igor Klatzo Conference proceedings 1983 Springer-V

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Morphofunctional Aspects of the Normal and Pathological Blood-Brain Barrier,ologist is often confronted with blood-brain barrier function since blood pigments appear in the cerebrospinal fluid in certain diseases. For example, in neonatal jaundice the yellow pigment reaches the orain because of an insufficiently developed blood-brain barrier; the phenomenon cannot be found in adults.
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Therapeutic Considerations in Blood-Brain Barrier Disturbances,viously, a most pertinent result of barrier damage occurring under clinical circumstances is vasogenic brain edema, acutely developing in head injury, focal ischemia, subacutely or chronically in brain tumours, brain abscess, inflammation, etc.
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Wentang Zhang,Yanbin Shen,Shaowu Niuion will be focused on microvessels (capillaries, precapillary arterioles and postcapillary venules) where extensive processes of exchange of various metabolites between blood and brain parenchyma take place.
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Pathophysiological Aspects of Blood-Brain Barrier Disturbances in Experimental Brain Tumors and Braissue hypoxia (5,6), and cellular uptake of extravasated serum proteins may induce reactive changes of the cell elements involved (7) . In the present investigation, some of these factors are discussed in two models of vasogenic brain edema: edema associated with experimental tumors, and edema assoc
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Morphofunctional Aspects of the Normal and Pathological Blood-Brain Barrier,concerned. This peculiarity has led to the concept of the so-called “blood-brain barrier” (BBB). The concept is useful not only when dealing with questions of metabolic exchange in general but more specifically for understanding the mechanism of the production of and the changes in the cerebral spin
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Morphology of Cerebral Endothelium and Astrocytes as Determinants of the Neuronal Microenvironment,art, to transport certain substrates while excluding other molecules because it comprises a continuous barrier that cannot be bypassed extra-cellularly. Each endothelial cell is joined to its neighbor by belts of tight junctions which occlude the intercellular clefts between them. In this way, the e
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