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Titlebook: Cerebral Blood Flow in Acute Head Injury; The Regulation of Ce Georg Emil Cold Book 1990 Springer-Verlag/Wien 1990 head.head injury.intracr

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0065-1419 (CMRO2), and intracranial pressure (ICP) in the acute phase after head injury have been published. These studies have been supplemented with studies of cerebral autoregulation, CO2 reactivity and barbiturate reactivity. Other investigations include studies of cerebrospinal fluid pH, bicarbonate, la
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Epidemiologie und Risikofaktorentension and hypertension may influence ICP, CBF, and CMRO.. Accordingly, studies in experimental models, including one or more of these pathogenic factors, are important in the understanding of the dynamic changes following head injury.
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The Effect of Barbiturate and Mannitol on CBF and Metabolism, severe head injury are available. On the other hand, barbiturate and mannitol have been used in the control of ICP-hypertension for many years. In the following the effects of barbiturate and mannitol on CBF and metabolism are reviewed.
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Summary,CO. and barbiturate (metabolic reactivity) in the acute phase after severe head injury and to discuss the therapeutical consequences with reference to prolonged artificial hyperventilation, hypothermia, barbiturate sedation, and mannitol therapy.
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Book 1990 The author‘s own clinical studies of the dynamic changes in CBF and cerebral metabolism are summarized and discussed, and the therapeutical implication as regards the use of artificial hyperventilation, sedation with barbiturate and mannitol treatment are discussed.
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