Titlebook: Cell Receptors; Morphological Charac G. Seifert Book 1991 Springer-Verlag Berlin Heidelberg 1991 Pathologie.Zellpathologie.cancer.cell.cell

VOK

8.1.1.1.3 Disubstituted phenoxyls,or these molecules can be divided into two general categories: (a) cell surface receptors for peptides, growth factors, and neurotransmitters that are linked to secondary events (some of which are still unknown) (. 1989), and (b) receptors for steroids, dihydrocholecalciferol, and thyroid hormones u

纵欲

8.1.1.1.3 Disubstituted phenoxyls,of thousands of interactions between living cells was later realized to be a fundamental mechanism of intercellular communication and a prerequisite for the functional integrity of multicellular organisms. Receptor proteins are essential (a) to transmit information from one cell to another, (b) to t

Hyaluronic-Acid

残忍

溃烂

MARS

8.1.1.1.3 Disubstituted phenoxyls, to postulate that specific cellular reactions to drugs must be based on so-called receptive substances. Receptors are proteins that specifically bind and take up hormones, growth factors, transport proteins, viruses, and toxins. The understanding of the physiological effects that are caused by cert

Debrief

8.1.1.1.3 Disubstituted phenoxyls,f the brain machinery at the microscopic and molecular levels. The broad applications, in the last three decades, of biochemical techniques to the study of nervous tissues has greatly increased our understanding of the chemical makeup of the brain. Because of the high regional and cellular complexit

CRATE

Disk199

8.1.1.1.3 Disubstituted phenoxyls,cortical hormones (glucocorticoids, mineralocorticoids), vitamin D, and insect hormones (ecdysteroids). As discussed in Sect. 3.1, these agents combine with intracellular receptor proteins, converting them to functional transcription factors, which then bind in the genome to influence the expression

博识

8.1.1.1.3 Disubstituted phenoxyls,plantability of malignant tumors in animals. The breakthrough became possible with the development of the techniques of DNA recombination, sequence analysis, and gene transfer. These methods made possible the isolation of the gene sequences responsible for the transforming potency of the Rous sarcom