Titlebook: Cell Impairment in Aging and Development; Vincent J. Cristofalo,Emma Holečková Book 1975 The Editor(s) (if applicable) and The Author(s),

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Time-Lapse Cinemicrophotographic Studies of Cell Division Patterns of Human Diploid Fibroblasts (WIes to study aging at the cellular level. The decline in proliferative capacity of diploid fibroblasts with increasing . age has been attributed to a gradual increase in the proportion of non-dividing cells (3,4,5,8).

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Alterations in Chromatin Functions during Aging ,, shown age-associated increases in glycogen content (1), lipid synthesis (2), lipid content (3), the number of lysosomes (4,5) and the specific activities of lysosomal enzymes (6–8). In addition, age related decreases in the specific activities of transketolase and 6-phosphogluconate dehydrogenase h

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Growth-Promoting Alpha-Globulin and Ageing,rm of dialyzed or undialyzed serum, Although some progress in identifying growth-promoting factors in serum has been made, the function of serum proteins in supporting the growth of cell cultures is not completely understood. In general, the growth-promoting properties of serum have been attributed

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,Cell Surface Alterations and “in Vitro” Aging of Animal Cells,otype. Conversely, abnormal cells survive indefinately (1). Several biochemical parameters have been studied in the past 10 years though very few of those studied have dealt with cell-surface integrity (2). However it is feasible to conceive that modifications of cell surface components would impair

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Changes in Lysosomes during Ageing of Parenchymal and Non-Parenchymal Liver Cells,tion and other changes occurring in the liver with age (1) will be reflected in metabolic changes at the cellular level. Since the liver consists of different cell types, a Particular ageing phenomenon observed for the intact organ or measured with a liver homogenate may be an expression of a genera