Titlebook: Cell Adhesion and Cytoskeletal Molecules in Metastasis; Anne E. Cress,Raymond B. Nagle Book 2006 Springer Science+Business Media B.V. 2006

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https://doi.org/10.1007/978-94-007-2762-5his chapter, the data from immunohistochemical analysis of human tissue and published DNA microarray results from human tissue are discussed to illustrate the altered expression of cell adhesion structures in carcinomas. Further, the role of adhesion structures in cancer metastasis is discussed and

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The Governance of Marine Resources,ss to metastasize. During prostate cacinogenesis, . gene expression or function is downregulated through multiple mechanisms, many of which combine to silence . expression through transcriptional regulation at the level of the . promoter. Recent evidence indicates that concomitant with the transcrip

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No-Till Agriculture in the USA, the necessary protrusions and forces that drive the cell forward. Several of the elements of the basic molecular machinery that assemble and operate the actin cytoskeleton have been identified and their function thoroughly characterized. Most of these elements are actin-binding proteins that can be

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https://doi.org/10.1007/978-94-007-4113-3s. We recently found that human EHM2 is androgen-regulated in a cancer cell line model of steroid-induced cytoskeletal reorganization, and expression profiling analyses by others have shown that it is a primary steroidregulated gene in rat liver and human lung cells. Bioinformatic analysis of human

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https://doi.org/10.1007/978-94-007-4156-0and contains, in part, the persistent basal cell population and the putative stem cells. It has been postulated by others that the transformation of normal glandular structures into cancer glands and subsequent tumor progression may involve aberrant regulation of the stem cell population. In this ch

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On the Biological Origin of Design in Natureenetics is a heritable change in phenotype that is independent of a change in the genotype. The focus of this chapter is to review, in part, the literature indicating that an “epigenetic switch” occurs in cancer progression and to detail the evidence of one particular gene product, maspin, as a para

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Liz Swan,Richard Gordon,Joseph Seckbachintegrin-mediated adhesion is sufficient to cause resistance to mechanistically distinct cytotoxics. This drug resistant phenotype is commonly referred to as cell adhesion mediated drug resistance or CAM-DR. The CAM-DR phenotype is observed in multiple tumor types that express a diverse array of onc