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派博传思国际中心 »期刊书目

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Titlebook: Cell Adhesion and Cytoskeletal Molecules in Metastasis; Anne E. Cress,Raymond B. Nagle Book 2006 Springer Science+Business Media B.V. 2006

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发表于 2025-3-21 19:22:17 | 显示全部楼层 |阅读模式
书目名称Cell Adhesion and Cytoskeletal Molecules in Metastasis
编辑Anne E. Cress,Raymond B. Nagle
视频videohttp://file.papertrans.cn/223/222731/222731.mp4
概述Compiled information on cell adhesion molecules in human cancer tissue specimens.Explains how cell adhesion influences DNA damage responses.Explains how cell adhesion influences chemotherapeutic drug
丛书名称Cancer Metastasis - Biology and Treatment
图书封面Titlebook: Cell Adhesion and Cytoskeletal Molecules in Metastasis; Anne E. Cress,Raymond B. Nagle Book 2006 Springer Science+Business Media B.V. 2006
描述.In this volume, the expression of specific adhesion molecules within human cancer tissues are highlighted. The expression signatures from published DNA microarray and immunohistochemistry studies are detailed. The concept that the alteration of specific adhesion molecules influence the cancer migration ability and cancer damage responses is detailed in this volume; both features are essential for the survival of an invading tumor cell. Defining the minimal adhesion receptors preserved on cancer cells during tumor progression will define the metastatic adhesion signature. Understanding the metastatic adhesion signature will reveal vulnerabilities that could be exploited for the prevention and/or eradication of the invading cancer cell..
出版日期Book 2006
关键词Migration; Regulation; adhesion structures; cancer; damage response; epithelium; extracellular matrix; tiss
版次1
doihttps://doi.org/10.1007/978-1-4020-5129-6
isbn_softcover978-90-481-7290-0
isbn_ebook978-1-4020-5129-6Series ISSN 1568-2102 Series E-ISSN 2215-1648
issn_series 1568-2102
copyrightSpringer Science+Business Media B.V. 2006
The information of publication is updating

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No-Till Agriculture in the USA,egulation of the actin cytoskeleton may be corrupted at several levels. This review examines several aspects of the actin cytoskeleton that may be affected during transformation and tumor progression.
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On the Biological Origin of Design in Naturetely silenced in cancer is discussed. Understanding the molecular features of the epigenetic switch is likely to provide a therapeutic opportunity to induce transcriptional reprogramming and prevent devastating disease processes such as epithelial cancer metastasis.
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On the Biological Origin of Design in Naturesurvival and growth response in DNA-damaged cells adherent to matrix proteins. Uncovering the molecular mechanisms may help to dissect central survival regulators in normal versus tumor tissue, which would provide the necessary knowledge for developing further innovative molecular-targeted anticancer strategies.
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CHANGING EXTRACELLULAR MATRIX LIGANDS DURING METASTASIS,oteins causing the release of growth factors or cryptic ECM peptides called matrikines. In this chapter, we will discuss changes that occur in ECM and examine how these changes are important for successful cancer metastasis.
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THE ACTIN CYTOSKELETON AND METASTASIS,egulation of the actin cytoskeleton may be corrupted at several levels. This review examines several aspects of the actin cytoskeleton that may be affected during transformation and tumor progression.
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CELL ADHESION-MEDIATED RADIATION RESISTANCE: THE ROLE OF INTEGRINS AND INTEGRIN PROXIMAL PROTEIN,survival and growth response in DNA-damaged cells adherent to matrix proteins. Uncovering the molecular mechanisms may help to dissect central survival regulators in normal versus tumor tissue, which would provide the necessary knowledge for developing further innovative molecular-targeted anticancer strategies.
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