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Titlebook: Carnitine Today; Claudio Simone,Giuseppe Famularo Book 1997 Springer Science+Business Media Dordrecht 1997 AIDS.T cell.biology.biosynthesi

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Effect of L-Carnitine on AZT-Induced Mitochondrial Toxicity: Studies on Human Muscle Cultures,tae was observed.. Molecular analysis has also determined a severe reduction of the mitochondrial DNA in the muscle of HIV patients treated with AZT.. We and others have reported on the mechanism by which AZT damages the muscle.. Our studies were performed in vitro, using human muscle cultures,. and
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National Spatial Planning in Chinae degradation of substrates represent the energy input and ATP the energy output. It must be emphasized that the actual energy currency of mitochondria is ΔμH on which, besides ATP synthesis, other relevant processes depend (i.e. metabolite and ion traffic through IMM).
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https://doi.org/10.1007/978-981-97-7729-7Fas stimulation by crosslinking with monoclonal antibodies induced peripheral blood T cell apoptosis in HIV-infected individuals.. In contrast, antibodies against other members of the TNF/NGF receptor family, such as CD27, CD30, CD40,4–IBB, p55 TNF receptor, p75 TNF receptor, and TNF receptor-relate
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Neue Trends zu G-CSF in der Onkologiein acylcarnitine (LCAC), which accumulates in ischemic cells.. The exchange may be catalyzed by a plasmalemmal translocase. In mitochondria such an exchange was proposed two decades ago. and has since been identified by Pande and Murthy in other organelles as well.. Indirect evidence forecast a simi
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