Titlebook: Cardiovascular Specific Gene Expression; Pieter A. Doevendans,Robert S. Reneman,Marc Bilsen Book 1999 Springer Science+Business Media Dord

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Peter Deuflhard,Andreas Hohmannaths were caused by cardiovascular disease and this is expected to rise to 36% in 2020. Incidence and ability to lower the number of cardiovascular deaths varies considerably from country to country. As shown by Sans et al. [2] standardized mortality from ischemic heart disease may vary in Europe fr

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https://doi.org/10.1007/978-3-030-73788-7th the actin-based cytoskeleton and the cell nucleus [1,2]. In the heart, MLP is expressed at high levels in atrial and ventricular myocytes during development and in the adult [1]. MLP consists of two LIM double-zinc fingers linked by a spacer of 58 residues. The LIM motif is a protein binding inte

HEED

Equations Governing Fluids in Motionctile and proliferative/ synthetic phenotypes. At present it is clear that such divisions are too simple. Vascular SMCs are in fact a collection of cells from various embryonic origins, with variations in morphology and gene expression patterns and with different functions [1,2,3,4,5]. Differences b

offense

https://doi.org/10.1007/978-3-030-73788-7entricular chambers. Cardiac sub-domains originate in early heart development: cardiomyocytes differentiate in an anterior-posterior gradient in the anterior lateral mesoderm prior to the formation of a transiently linear heart tube. Studies in the chick have shown that the cardiac tube contains fut

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Surface Charge Simulation Method (SSM)of families with cardiac diseases like hypertrophic cardiomyopathy and dilated cardiomyopathy (DCM). Although four chromosomal loci are associated with the inherited form of DCM, the genes involved are still unknown [1]. In most patients with dilated cardiomyopathy inheritance appears less important