Titlebook: Cardiovascular Specific Gene Expression; Pieter A. Doevendans,Robert S. Reneman,Marc Bilsen Book 1999 Springer Science+Business Media Dord

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书目名称Cardiovascular Specific Gene Expression影响因子(影响力)




书目名称Cardiovascular Specific Gene Expression影响因子(影响力)学科排名




书目名称Cardiovascular Specific Gene Expression网络公开度




书目名称Cardiovascular Specific Gene Expression网络公开度学科排名




书目名称Cardiovascular Specific Gene Expression被引频次




书目名称Cardiovascular Specific Gene Expression被引频次学科排名




书目名称Cardiovascular Specific Gene Expression年度引用




书目名称Cardiovascular Specific Gene Expression年度引用学科排名




书目名称Cardiovascular Specific Gene Expression读者反馈




书目名称Cardiovascular Specific Gene Expression读者反馈学科排名

Arctic

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Acetaminophen

Equations Governing Fluids in Motion of SM22a have been described that make this gene a promising candidate [3,10]. Marker proteins are not only important as molecules that allow a better definition of SMC variants, but also because they may provide promoter sequences that can be used as instruments to manipulate gene expression in SM

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https://doi.org/10.1007/978-3-030-73788-7t growth, septation, and remodelling of these distinct cardiac segments result in the development of separate right and left atrial and ventricular chambers with independent inlets and outlets. Morphological remodelling between different cardiac sub-domains is often perturbed in congenital heart def

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Seismic Analysis of Pine Flat Concrete Damyocardium. However, during development the predominant isoform expressed is slow skeletal troponin I [2]. We have previously documented aspects of troponin expression in the human heart [3–6] and demonstrated a developmental switch in troponin I expression during human development. Analysis of mRNA

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Optimizing Electromagnetic DevicesAV-node, the top of the AV-bundle and the top of the bundle branches. This is true for both neonatal and adult rat heart. By contrast, Cx40 exibits a clearly different expression pattern [2]. During embryonic development, Cx40 expression is widespread throughout the myocardium, but is gradually down

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Variational Finite Element Methodf inherited cardiac arrhythmias: the Long QT Syndrome (LQTS) [1] and the Brugada Syndrome [2]. In the cases of these diseases identified mutations in ion channel structure have been shown to cause functional changes in channel properties using heterologous expression of the channel proteins [3]. The

GNAT

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