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Titlebook: Cardiovascular Complications in Cancer Therapy; Antonio Russo,Giuseppina Novo,Fausto J. Pinto Book 2019 Springer Nature Switzerland AG 201

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发表于 2025-3-21 19:23:06 | 显示全部楼层 |阅读模式
书目名称Cardiovascular Complications in Cancer Therapy
编辑Antonio Russo,Giuseppina Novo,Fausto J. Pinto
视频video
概述Provides a comprehensive yet concise resource..Provides a guide for the clinician facing cardiovascular toxicity in cancer and hematologic patients..Written by experts in the field.
丛书名称Current Clinical Pathology
图书封面Titlebook: Cardiovascular Complications in Cancer Therapy;  Antonio Russo,Giuseppina Novo,Fausto J. Pinto Book 2019 Springer Nature Switzerland AG 201
描述This proposed text is designed to provide a useful and comprehensive resource and state-of-the-art overview to readers about vascular damage  potentially induced by antineoplastic drugs. .Thanks to more and more effective antineoplastic treatments the survival of cancer patients is enormously increasing, but at the same time it is increasing the burden of related cardiovascular complications that affect morbidity and mortality. On this basis a new branch of cardiology has been developed, that is Cardio-Oncology. The aim is to prevent cardiovascular complications related to cancer therapy and to facilitate and avoid interruption of antineoplastic drugs due to the occurrence of cardiovascular damage. An increasing attention has been given to cardiac damage, while, until today, vascular complications have been poorly evaluated. .The aim of this book is to focus on vascular complications related to cancer treatment, to guide the clinician at facing,during his every day practice, cardiovascular toxicity in cancer and hematologic patients. .The proposed sections of the book have been structured to review the molecular mechanisms underlying vascular damage induced by new and old treatment
出版日期Book 2019
关键词anti HER-2 inhibitors; anti-VEGF therapy; Pulmonary hypertension; Venous thromboembolism; Peripheral art
版次1
doihttps://doi.org/10.1007/978-3-319-93402-0
isbn_softcover978-3-030-06653-6
isbn_ebook978-3-319-93402-0Series ISSN 2197-781X Series E-ISSN 2197-7828
issn_series 2197-781X
copyrightSpringer Nature Switzerland AG 2019
The information of publication is updating

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Heart Failure and Left Ventricular Dysfunction,cause is, are to improve their clinical status, functional capacity, and quality of life, prevent hospital admission, and reduce mortality. However, the real challenge of cardio-oncology is to avoid that damage occurs at all..For this reason early diagnosis of myocardial impairment is crucial, and i
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Peripheral Artery Disease and Stroke,longer time of drug exposure, and sequential use of potent TKIs. In addition, circulating concentrations of VEGF are reduced by cyclophosphamide administered at continuous low doses, which might underpin some of the observed vascular toxicity, as seen in patients treated with VEGFIs. This alkylating
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Atrial Fibrillation in Cancer,cally every form of cardiac or vascular disease. Arrhythmias represent a significant part of this spectrum, and atrial fibrillation (AF) is one of the main arrhythmias occurring in cancer patients..Several studies have documented the relationship between cancer and AF. In terms of epidemiology, AF i
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Diagnosis of Cardiac Damage: Role of Stress Echo,motherapy, a complete evaluation of systolic and diastolic function at rest and after stress should be performed. Contractile reserve assessed by either ejection fraction or speckle tracking echocardiography should be determined considering its diagnostic and prognostic power. New technologies appli
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Early Detection and Monitoring of Vascular Damage, Accordingly, promptly detection of these possible adverse effects by vascular imaging should be strongly promoted to introduce efficient prophylactic therapies when indicated. It is fundamental to diagnose vascular complications and treat them aggressively in order to reduce deleterious adverse eff
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Prevention and Clinical Management of Cardiovascular Damage Induced by Anticancer Drugs: Need for Etion of a large number of molecular features and have the potential to identify new factors that contribute to cardiac and endothelial function and how they interact. These technologies could play a pivotal role in unraveling the pathophysiology of vascular damage induced by anticancer treatment, in
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