Titlebook: Cardioprotection Against Acute Myocardial Infarction; Shinya Minatoguchi Book 2019 Springer Nature Singapore Pte Ltd. 2019 Acute myocardia

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https://doi.org/10.1007/978-981-99-5281-6 and oxytocine are effective in reducing the infarct size and improving the cardiac function when administered after the onset of AMI. We clarified the mechanisms of how these drugs have beneficial effects.

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Metagenomics for Drug Discoverynesis and activating prosurvival signals. The combination of G-CSF and erythropoietin-encapsulated liposomes with SLX was more cardiprotective than G-CSF or EPO-liposome with SLX alone. In vivo hepatocyte growth factor (HGF) gene transfer reduced myocardial ischemia-reperfusion injury through its multiple actions.

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Pharmacological Preconditioning,s, β-blockers, HMG-CoA reductase inhibitor, anti-platelets, ARBs, and ACE inhibitors show pharmacological preconditioning effects on the myocardial infarct size and cardiac function, and the precise mechanisms leading to the beneficial effects of these drugs are discussed.

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