Titlebook: Cardiac-Vascular Remodeling and Functional Interaction; Yukio Maruyama (Professor and Chairman),Masatsugu Book 1997 Springer Japan 1997 a

malign

Transient Hemodynamic Perturbations: An Approach to the Study of Ventricular Interaction and Restoriem, we have developed approaches using transient (single-beat) hemodynamic perturbations that eliminate the confounding effects of series interactions and filling variations. In experimental studies in open-chest dogs, we have employed rapid changes in right heart volume to quantitate direct right-t

议程

Extrinsic Versus Intrinsic Determinants of the Diastolic Pressure—Volume Relationxternal to the heart, principally resulting from pericardial and right heart loads. Intrinsic factors can be divided into passive and active components. Passive elements include myocardial viscoelasticity, caused by structural proteins within the sarcomere, extra-cellular matrix proteins, and corona

天空

Endocardial—Myocardial Interactionxistence of an endocardium-mediated intracavitary autoregulation of cardiac performance has been postulated. Recent discoveries on morphology and function of the endocardium have substantiated this hypothesis. ., phenotypic expression of several receptors, tight and gap junctions, adhesion molecules

Evolve

Arterial Compliance as Load on the Heartperiod), these are the major determinants of systolic and diastolic pressure in the ascending aorta. A decrease in total arterial compliance will, in the nonregulated cardiovascular system, decrease cardiac output and thus mean pressure while increasing pulse pressure. The result is a large decrease

Stress-Fracture

Optimal Afterload that Maximizes External Work and Optimal Heart that Minimizes O2 Consumptionmaximizes external work. A theoretical analysis indicated, for a given end-diastolic volume, that external work becomes maximal when effective arterial elastance (..) equals end-systolic elastance (..). The ratio of external work to its maximum value was used to indicate the optimality of the afterl

arcane

Influence of Aortic Impedance on the Development of Pressure-Overloaded Left-Ventricular Hypertrophy hypertrophy. To test whether the sustained change in aortic impedance might affect the mode of the development of LV hypertrophy, coarctation of either the ascending (group 1) or abdominal aorta (group 2) was performed in 6-week-old rats. Four weeks after the operation, both groups developed LV hyp

矛盾心理

https://doi.org/10.1007/978-4-431-67041-4adaptation; artery; cardiac function; cardiovascular; circulation; endothelium; heart; heart disease; heart

defendant

Respiratory therapy for children,n-to eightfold. This is accomplished by acute increases in heart rate and stroke volume. The elevations in stroke volume are produced through coordinated adjustments to the cardiac, respiratory, and peripheral circulatory systems which result in 1) enhanced myocardial contractility, 2) decreased sys

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,Nursing in Times of Socialism (1970–1973),changes in cardiac muscle function, heart failure is associated with alterations in the vasculature. Functional and structural alterations in the coronary circulation, including a failure of adaptive growth of the coronary circulation, may contribute to the loss of muscle function of the heart and m

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