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Titlebook: Cardiac-Vascular Remodeling and Functional Interaction; Yukio Maruyama (Professor and Chairman),Masatsugu Book 1997 Springer Japan 1997 a

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发表于 2025-3-21 17:05:33 | 显示全部楼层 |阅读模式
书目名称Cardiac-Vascular Remodeling and Functional Interaction
编辑Yukio Maruyama (Professor and Chairman),Masatsugu
视频videohttp://file.papertrans.cn/222/221846/221846.mp4
图书封面Titlebook: Cardiac-Vascular Remodeling and Functional Interaction;  Yukio Maruyama (Professor and Chairman),Masatsugu  Book 1997 Springer Japan 1997 a
描述Cardiac performance is regulated not only by cardiac muscle properties but also by several other factors, including those associated with the neurohumoral system and the mechanical characteristics of the peripheral circulation. New information con­ cerning these regulatory factors has furthered our understanding of the pathophysi­ ology of cardiac dysfunction. However, controversy remains, along with a need to integrate these multidisciplinary findings. It was with this in mind, together with my continuing interest in the response of the normal and diseased heart to variations in loading conditions, that the satellite symposium entitled "Interactions Between Car­ diac Function and Vascular Dynamics" was organized and dedicated to my mentor, Dr. T. Takishima. The symposium was held in Fukushima, Japan, in 1992 following the Tenth Inter­ national Conference of the Cardiovascular Systems Dynamics Society in Kobe, Japan, which was organized by the then president of the society, Dr. Masatsugu Hori. The Fukushima symposium and the Kobe conference were stimulating and informative. To commemorate these events, Dr. Hori, Dr. Janicki, and I decided to publish this book. It covers topics that
出版日期Book 1997
关键词adaptation; artery; cardiac function; cardiovascular; circulation; endothelium; heart; heart disease; heart
版次1
doihttps://doi.org/10.1007/978-4-431-67041-4
isbn_softcover978-4-431-67043-8
isbn_ebook978-4-431-67041-4
copyrightSpringer Japan 1997
The information of publication is updating

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发表于 2025-3-21 20:30:43 | 显示全部楼层
Vascular Remodeling During Heart Failurechanges in cardiac muscle function, heart failure is associated with alterations in the vasculature. Functional and structural alterations in the coronary circulation, including a failure of adaptive growth of the coronary circulation, may contribute to the loss of muscle function of the heart and m
发表于 2025-3-22 00:53:02 | 显示全部楼层
Myocardial Interstitial Collagen Matrix Remodeling in Response to a Chronic Elevation in Ventricularindividual myocytes, groups of cardiac myofibrils, muscle fibers, and muscle fiber bundles. Accordingly, it provides for cardiac myocyte and muscle fiber alignment, and in so doing it imparts a mechanical support to the myocardium that assists in the maintenance of ventricular shape, size, and funct
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Cardiac Hypertrophy and the Renin-Angiotensin Systemx sequence of compensatory events that ultimately result in an adversely remodeled myocardium and a dilated, thin-walled, spherical ventricle. For a period of time, a preclinical heart failure state may exist in which there is ventricular dysfunction caused by myocardial damage but no clinical evide
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The Role of Manganese Superoxide Dismutase in the Acquisition of Tolerance of the Heart to Ischemia:olerance acquisition from the aspect of oxygen radical metabolism in the heart. Manganese superoxide dismutase (Mn-SOD), which scavenges the initial reductive metabolite of molecular oxygen on ischemia-reperfusion, was induced after various external stimuli including heat shock, α.-adrenergic stimul
发表于 2025-3-22 20:56:41 | 显示全部楼层
Remodeling: How Vessels Narrowmarkable ability of normal vessels to maintain the caliber appropriate for the blood flow needed for the subservient organ. Indeed, the ability to adjust caliber must be very primitive. The vessel tree, like any closed circuit, can only continue to conduct blood if each branch has an appropriate res
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Interaction Between Left-Ventricular Function and Loading Condition in Cardiovascular Diseaseenesis of the progression or amelioration of cardiac dysfunction. A decrease in peripheral resistance increases stroke volume until a certain critical level of afterload reduction; thus, afterload-reducing therapy is frequently performed in patients with heart failure. When afterload reducing is per
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