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Titlebook: Cardiac Cytoarchitecture; How to Maintain a Wo Elisabeth Ehler Book 2015 Springer International Publishing Switzerland 2015 Actin and Myosi

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书目名称Cardiac Cytoarchitecture
副标题How to Maintain a Wo
编辑Elisabeth Ehler
视频video
概述Gives a concise and comprehensive overview of cardiac cytoarchitecture.Treats the different subcellular compartments of the cardiomyocyte.Addresses development, disease and basic cell biological quest
图书封面Titlebook: Cardiac Cytoarchitecture; How to Maintain a Wo Elisabeth Ehler Book 2015 Springer International Publishing Switzerland 2015 Actin and Myosi
描述.This book presents a collection of expert reviews on different subcellular compartments of the cardiomyocyte, addressing fundamental questions such as how these compartments are assembled during development, how they are changed in and by disease and which signaling pathways have been implicated in these processes so far. As such, it offers the first overview of the cell biology of heart disease of its kind, addressing the needs of cell biology students specializing in vascular and cardiac biology, as well as those of cardiologists and researchers in the field of cell biology..
出版日期Book 2015
关键词Actin and Myosin in the heart cell; Cardiac cell biology; Cardiomyocte; Cardiomyopathy; Heart cell ultra
版次1
doihttps://doi.org/10.1007/978-3-319-15263-9
isbn_softcover978-3-319-34653-3
isbn_ebook978-3-319-15263-9
copyrightSpringer International Publishing Switzerland 2015
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Ca2+ Regulation of the Cardiac Thin Filament,ltimately allow the force generation required for the maintenance of normal cardiac pump function. This chapter seeks to delve into the biochemistry and biophysics that dictate the cellular physiology of the myofilaments.
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https://doi.org/10.1007/978-3-540-68430-5 the ID is presented. Because of its complex nature, it is not surprising that the ID has been implicated in a number of heart diseases and malfunctions. Changes in its structure and composition associated with these heart problems are described with special regard to the role of the ID in dilated cardiomyopathy.
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