Titlebook: Cardiac Amyloidosis; Diagnosis and Treatm Michele Emdin,Giuseppe Vergaro,Marianna Fontana Book 2024 The Editor(s) (if applicable) and The A

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Self-Scheduling MPC Using LPV Modelsmyocardial texture assessed by native T1 or T2 can help differentiate CA from other forms of left ventricular hypertrophy. Finally, some AI models (cluster analyses) have been associated with the outcome.

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s to be a timely one, given the great interest of clinicians and researchers in CA and the exponential increase in publications in recent year,and will be of interest to cardiologists, internal medicine an978-3-031-51759-4978-3-031-51757-0

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A Brief History of Amyloidosis,lia with apple green birefringence under polarized light was the first diagnostic criterion for amyloid, and it is still relevant for diagnosis. From the second half of the twentieth century, electron microscopy allowed to characterize the ultrastructure of amyloid deposits. Furthermore, a growing n

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Pathophysiology, Classification, and Epidemiology of Amyloidosis,ns, serum amyloid P component) that are believed to stabilize amyloid deposits and promote further deposition of amyloidogenic proteins. Once considered a rare disease, it is now well acknowledged that, depending on the amyloid precursors, the prevalence of amyloidosis may be frequent, at least in s

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Amyloid Light Chain (AL) Amyloidosis,ove and type the amyloid deposits, determine the monoclonal protein and free light chains, and assess the disease stage with natriuretic peptides and troponin. In those with monoclonal gammopathies of uncertain significance or smoldering myeloma, or when heart failure symptoms are present, cardiac b

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Hereditary Transthyretin Amyloidosis,tations, being also influenced by sex, ethnicity, and still putative geographical and environmental factors. ATTRv-PN typically presents with progressive length-dependent sensorimotor polyneuropathy, initially affecting lower limbs, and autonomic neuropathy. ATTRv-CM main manifestations are heart fa

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Wild-Type ATTR Amyloidosis,duction disturbances. The musculoskeletal soft tissues may be infiltrated early, on average 5–15 years before cardiac manifestations. The most frequent orthopedic manifestations include: carpal tunnel syndrome, brachial biceps tendon rupture, lumbar spinal stenosis, joint replacement (hip, knee, and

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Plasma Transthyretin and Its Ligands,ion of genetic variants. Furthermore, the influence of T4 and holoRBP4 on TTR structural stability will be discussed. Further studies are needed to understand whether and to what extent T4 and holoRPB4 can influence the course of wild-type or variant ATTR.