Titlebook: Cardiac Allograft Rejection; G William Dec,Jagat Narula (Director),Ignasi Carri Book 2001 Springer Science+Business Media New York 2001 Im

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Immunology of Cellular and Humoral Rejection after Cardiac Transplantation cardiac transplantation that serum samples from these patients contained antibodies directed against components of cardiac myofibres. These antibodies were detected early (4 or 5 days) after transplantation and their presence appeared to correlate with clinical signs of rejection.

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Evolution of Noninvasive Testing for the Detection of Cardiac Allograft Rejectione multiple invasive procedures. Moreover, endomyocardial biopsy may not predict the occurrence of clinically significant acute allograft rejection ina small number of patients. Accordingly, the search for new and noninvasive techniques for the diagnosis of cardiac allograft rejection has continued to receive much interest

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Book 2001an unproven experimental surgicaltechnique to the most effective form of therapy for refractoryend-stage heart disease. It has captured the public‘s imagination andexpanded our understanding of fundamental immunologic mechanisms thatare responsible for cellular and humorally-mediated immunity. Despi

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Immunobiology of Chronic Cardiac Allograft Rejectionracic organ transplantation has increased constantly over the past decade as a result of new surgical techniques, immunosuppressive protocols, and innovations in managing acute rejection and infection, particularly cytomegalovirus (CMV) infection. Despite the substantial improvement in early surviva

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Inflammatory Cell Infiltration, Cytokines, and Mechanisms of Myocyte Necrosis in Cardiac Transplant iac tissue, the major lineage being highly specialized, terminally differentiated parenchymal cardiac myocytes. The traditional approach to optimizing survival of the allograft has been to target what have been considered as immune-mediated mechanisms of rejection. However, it is now becoming increa

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Apoptosis in Cardiac Transplant Rejection mandatory for tissue development and growth in order to maintain the balance between new and old cells. Contrary to necrosis, apoptosis is an active energy-requiring process and is genetically programmed (Kerr 1972). Ultrastructural characterization of the process of apoptosis reveals that it invol

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