Titlebook: Captopril and Hypertension; David B. Case,Edmund H. Sonnenblick,John H. Laragh Book 1980 Springer Science+Business Media New York 1980 blo

lethargy

Lacerate

Working-Memory

Captopril (Capoten®; SQ 14,225) (,-3-Mercapto-2-methylpropanoyl-,-proline)-converting enzyme (ACE) [E.C. 3.4.15.1] has also been designated as converting enzyme (CE), peptidyldipeptide carboxy hydrolase, kininase II, or “bradykininase.” Hence, in vivo inhibition of ACE will reduce the pressor activity of AI, but not that of AII, and augment the vasodepressor activity of bradykinin (BK).

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Toxicologic and Drug Metabolic Studies of SQ 14,225 in Animalsbin et al. in other chapters in this volume. This chapter will delineate the results of a number of studies that characterized the acute and subacute toxicities of SQ 14,225 in laboratory animals. Some metabolic data from preliminary studies in rats and dogs are also included.

SEVER

Blood Pressure Homeostasisrious organ systems in amounts appropriate to their needs is accomplished by constriction or relaxation of vascular smooth muscle. Within this closed system, a pressure gradient is discerned, with pressure greatest in the aorta and lowest in the vena cavae.

Gustatory

Mechanisms of Hypertension Induced by Electrolyte-Active Steroidssterone acetate-saline (DOCA-saline) hypertensive rat. may have little clinical relevance because, in this model, hypertension is induced by reducing renal mass and by the administration of large amounts of salt together with DOCA. On the other hand, clinical studies in the syndrome of primary aldos

BLA

–DOX

The Influence of Various Neurological Defects on the Release of Renin in Normal Mandifference between the rise of plasma renin activity on standing compared with the slower increase under the influence of sodium deprivation. In examining the situation in normal man, it is important to try to incorporate the findings from various other types of experimental studies, ranging from th

中止

Synovial-Fluid

Systemic Vascular Resistance The smooth muscle tone of these vessels is influenced by neural, humoral, and local environmental factors. In addition, changes in the caliber of these vessels may result from structural alterations, including the accumulation of water and electrolytes in the walls. The tone of these vessels also m