Titlebook: Calcium in Drug Action; George B. Weiss Book 1978 Plenum Press, New York 1978 Prostaglandin.calcium.drug.metabolism.pharmacology.research

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https://doi.org/10.1007/978-3-531-90169-5y, such as the ion gradients across nerve membranes, and is initiated by removal of inhibition; and (4) the information transfer frequently involves amplification (multiple copies of a template, many product molecules generated by one molecule of activated enzyme, etc.).

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https://doi.org/10.1007/978-3-531-91788-7hich have attempted to define the various mechanisms of Ca. regulation in this tissue. To date, no unifying hypothesis can be drawn regarding the mechanism(s) of regulation as can be done for other muscle tissue.

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Surface Calcium in the Heart: Its Function and Role in Drug Action concentration (Rich and Langer, 1975; Lüllmann .., 1974). When a skeletal muscle is perfused with a Ca.-free medium the decline of tension has a T. of 23 min (Fig. 1). The large lateral sacs of the sarcoplasmic reticulum serve in skeletal muscle as storage and release sites for activator Ca. (Endo and Nakajima, 1973; Winegrad, 1968).

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Book 1978current interest. The scope of investi­ gated actions and reactions in which a role for calcium ion is of some importance is so numerous as to convey the impression that calcium ion is everywhere and interacts with everything. This being so, the challenge in contemporary research is to focus on inve

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https://doi.org/10.1007/978-3-531-91788-7d no additional depolarization (Thesleff, 1955). Under these conditions, the end-plate membrane receptors gradually became refractory to agonist action and were said to be “desensitized” (Thesleff, 1955).

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