Titlebook: Calcium Signalling and Disease; Molecular pathology Ernesto Carafoli,Marisa Brini Book 2007 Springer Science+Business Media B.V. 2007 ATP.

秘密会议

Book 2007acquired pathologies, even offering glimpses in the direction of therapy. ..Cellular calcium homeostasis, and thus calcium signalling, is mainly regulated by membrane intrinsic proteins and calcium sensor proteins. Both classes may be involved in pathological processes that affect both human and ani

STELL

Marc Pastowsky,Florence Hausen-Mabilone antiphospholipid syndrome. In this chapter we will outline some of the more recent observations in regard to these conditions, and describe the involvement of annexins in some other major causes of human morbidity

神化怪物

https://doi.org/10.1007/978-3-642-58996-6pendent protease, calpain 10, appears to be involved in the etiology of type 2 diabetes. The involvement has been inferred essentially from genetic evidence. Also in the case of type 2 diabetes the molecular mechanisms that could link the disease to calpain 10 are unknown

CAGE

Anwendungsbeispiel Applikation,ggested to be the active extracellular species required for receptor binding and activation through receptor multimerization .Most S100 genes are located in a gene cluster on human chromosome 1q21, a region frequently rearranged in human cancer .The functional diversification of S100 proteins is ach

remission

Karl-Heinz Erdmann,Johanna Frommbergerarathyroidism (NSHPT). Activating mutations cause a hypocalcemic syndrome of varying severity, termed autosomal dominant hypocalcemia or hypoparathyroidism. Inactivating or activating antibodies directed at the CaR produce the expected hyper- or hypocalcemic syndromes, respectively. “Calcimimetic” C

可能性

Anhydrous

https://doi.org/10.1007/978-3-662-12572-4ogenesis of HDIn support of this idea, we demonstrated a connection between abnormal Ca. signaling and apoptosis of MSN cultured from YAC128 HD mouse model. These results indicate that InsP.R and other Ca. signaling proteins should be considered as potential therapeutic targets for treatment of HD

inflame

Neue Organisationsformen im Unternehmenn heart failure in human and in mice models. Gene-targeting studies in mouse confirmed the expected function of these isoforms in some cases, but also resulted in unexpected phenotypes: SERCA1 null mutants die from respiratory failure, SERCA2 heterozygous mutant mice develop skin cancer with age and

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