Titlebook: CD95; Methods and Protocol Patrick Legembre Book 2017 Springer Science+Business Media LLC 2017 Cell motility.Immunoprecipitation.TNF Recept

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书目名称CD95影响因子(影响力)




书目名称CD95影响因子(影响力)学科排名




书目名称CD95网络公开度




书目名称CD95网络公开度学科排名




书目名称CD95被引频次




书目名称CD95被引频次学科排名




书目名称CD95年度引用




书目名称CD95年度引用学科排名




书目名称CD95读者反馈




书目名称CD95读者反馈学科排名

造反,叛乱

zonules

Immunoprecipitation of Death Inducing Signaling Complex by Caspase-8,ation is an elegant and efficient procedure to investigate endogenous protein interactions or complexes. Provided that the targeted complex is soluble in mild detergent these complexes can be recovered using protein A/G-coupled Sepharose beads and further analyzed after denaturation and electrophore

字的误用

In Vitro Evaluation of the Apoptosis Function in Human Activated T Cells, the intrinsic pathway on in vitro activated T cells. Her, we describe an in vitro assay allowing the monitoring of three different apoptosis pathways: (1) the FAS-induced pathway, (2) the activation-induced cell death (AICD), and (3) the death induced by starvation of the cells, called activated-ce

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Proximity Ligation Assay (PLA) to Evaluate DISC and MISC Composition,c signal at the detriment of the non-apoptotic signaling pathway [1–6]. Also CD95 can form different protein complexes via dynamic protein–protein interactions (PPIs) according to its interaction with soluble or transmembrane CD95L. Therefore, spatiotemporal identification of these PPIs is pivotal t

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不朽中国

armistice

CD95-Mediated Calcium Signaling,r calcium signals that control the cell fate, apoptosis, or survival, depending on the ligand (membrane or soluble). Intracellular calcium determination is an exquisite readout to explore the molecular mechanisms elicited by CD95 engagement. The most widely applied methods for studying calcium signa

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南极

Study of the CD95-Mediated Non-apoptotic Signaling Pathway: PI3K,ence 245(4915):301–305, 1989). Accumulating evidence indicate that this so-called death receptor can also trigger non-apoptotic signaling pathways promoting inflammation and oncogenesis (Barnhart et al., Embo J 23(15):3175–3185, 2004; Chen et al., Nature 465(7297):492-496, 2010; Legembre et al., Cel