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Titlebook: Breathing, Feeding, and Neuroprotection; Ikuo Homma (Professor and Chairman),Seiji Shioda ( Conference proceedings 2006 Springer-Verlag To

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Management der digitalen Transformationnergics, which simulate transport, and triglycerides, which inhibit it. The ability of triglycerides to inhibit leptin transport may have evolved as a response to the hypertriglyceridemia of starvation. If so, their ability to inhibit leptin transport in obesity may be a metabolic case of mistaken identity.
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Management der frühen Innovationsphasenocytes even at a concentration below 2 nM. These results indicate that PACAP at a low concentration prefers induction of differentiation into astrocytes rather than proliferation in neural stem cells.
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Management der frühen Innovationsphasenat 100 or 300 mg/kg did not induce cell death in the subventricular zone (SVZ) of the lateral ventricle, known to be a site of adult neurogenesis. These findings strongly suggest that 1) sensitivity to the genotoxicity differes among fetal neural stem cells, 2) BrdU-labeling should be avoided when early neurogenesis is being observed.
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Management der frühen Innovationsphasenine functions. Hypocretin-deficient narcolepsy thus appears now to be a more complex condition than a simple sleep disorder, and it may serve a disease model to study the most fundamental hypothalamic function.
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Differentiation of Neural Stem Cells into Astrocytes by Low Concentration of Pituitary Adenylate Cycocytes even at a concentration below 2 nM. These results indicate that PACAP at a low concentration prefers induction of differentiation into astrocytes rather than proliferation in neural stem cells.
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Neurogenesis is Toxicologically Heterogeneous: A Case of BrdU-lnduced Cell Deathat 100 or 300 mg/kg did not induce cell death in the subventricular zone (SVZ) of the lateral ventricle, known to be a site of adult neurogenesis. These findings strongly suggest that 1) sensitivity to the genotoxicity differes among fetal neural stem cells, 2) BrdU-labeling should be avoided when early neurogenesis is being observed.
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Hypocretin-Deficient Narcolepsy as a Disease Model to Study the Hypothalamic Function in Health and ine functions. Hypocretin-deficient narcolepsy thus appears now to be a more complex condition than a simple sleep disorder, and it may serve a disease model to study the most fundamental hypothalamic function.
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Heiko von der Gracht,Stefanie Kisgenbrainstem and spinal cord of 0- to 1-day-old Wistar rats and 0-day-old mice isolated under deep ether anaesthesia were incubated in a modified Krebs solution containing a fluorescent voltage-sensitive dye. Fluorescence signals corresponding to respiratory activity were detected by a CCD image sensor
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