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Titlebook: Breast Cancer: Cellular and Molecular Biology; Marc E. Lippman,Robert B. Dickson Book 1988 Kluwer Academic Publishers, Boston 1988 Staging

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Takahiro Otabe,Yuta Nihongaki,Moritoshi Satohe various factors participate in the etiology of breast cancer have been confounded by a lack of information on specific genetic mutations associated with the initiation or progression of the disease. Three general approaches have been used to attempt to identify genetic mutations associated with breast cancer.
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https://doi.org/10.1007/978-3-642-64993-6y because of their oncogenic activity, offer an additional utility as models of gene regulation. When integrated in the host cell chromosome, these viruses adopt a low copy template structure (provirus) more similar to host cell genes than that of the high copy DNA viruses.
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F. Peter Guengerich,Francis K. Yoshimotont earlier discoveries on MMTV will be summarized to provide a framework of the biology of viral mammary tumorigenesis. More encyclopedic overviews of the biology and molecular biology of MMTV are found in references 1, 2 and 3.
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Takahiro Otabe,Yuta Nihongaki,Moritoshi Satoconditions. Since recourse to . studies is not possible with humans, the fidelity with which the culture system reflects the . situation is particularly important, if the information obtained . is to have meaning for understanding the normal and perturbed functions of the human breast. We describe h
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Takahiro Otabe,Yuta Nihongaki,Moritoshi Satoe physiological status of the host [1, 2]. As a consequence, several different types of breast tumors can be distinguished by histopathological criteria, chromosomal abnormalities, hormone receptor status, and other biochemical characteristics. However, attempts to derive a cohesive picture of how t
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Akihiro Eguchi,Masahiro Kawaharaly conserved in vertebrate evolution. Acute transforming retroviruses have substituted viral genes essential for replication with these discrete segments of host genetic information. When incorporated within the retroviral genome, such transduced sequences acquire the ability to induce neoplastic tr
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