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Titlebook: Breast Cancer: Cellular and Molecular Biology; Marc E. Lippman,Robert B. Dickson Book 1988 Kluwer Academic Publishers, Boston 1988 Staging

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发表于 2025-3-21 19:35:42 | 显示全部楼层 |阅读模式
期刊全称Breast Cancer: Cellular and Molecular Biology
影响因子2023Marc E. Lippman,Robert B. Dickson
视频video
学科分类Cancer Treatment and Research
图书封面Titlebook: Breast Cancer: Cellular and Molecular Biology;  Marc E. Lippman,Robert B. Dickson Book 1988 Kluwer Academic Publishers, Boston 1988 Staging
影响因子Where do you begin to look for a recent, authoritative article on the diagnosis or management of particular malignancy? The few general oncology text­ books are generally out of date. Single papers in specialized journals are informative but seldom comprehensive; these are more often preliminary reports on a very limited number of patients. Certain general journals fre­ quently publish good in-depth reviews of cancer topics, and published sym­ posium lectures are often the best overviews available. Unfortunately, these reviews and supplements appear sporadically, and the reader can never be sure when a topic of special interest will be covered. Cancer Treatment and Research is a series of authoritative volumes which aim to meet this need. It is an attempt to establish a critical mass of oncology literature covering virtually all oncology topics, revised frequently to keep the coverage up to date, easily available on a single library shelf or by a single personal subscription. We have approached the problem in the following fashion. First, by divid­ ing the oncology literature into specific subdivisions such as lung cancer, genitourinary cancer, pediatric oncology, etc. Second, by a
Pindex Book 1988
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Peter Blazso,Ildiko Sinko,Robert L. Katonan the secondary structure [3]. Mouse EGF is produced from a much larger precursor molecular of 130,000 Daltons [4]. In the mid 1970s, EGF was detected [5] and isolated from human urine [6]. In 1975 Gregory demonstrated that human EGF was equivalent to urogastrone, a hormone capable of inhibiting gas
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Sara Gil-Perotin,Patrizia Casaccia-Bonnefilaffect mammary tumor cell growth? Cell lines clearly have the advantage of relative cellular homogeneity, reproducibility from one experiment to another, ease of experimental manipulation and an approximately defined medium in which growth can be studied. The word ‘approximately’ is used because, al
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Promoter Activation with Electromagnetismnd as products of discrete cell types acting as either paracrine or autocrine factors [2–6]. This raises a second important point, namely that the mammary gland is not homogeneous but is a heterogenous tissue in which the mammary epithelial and myoepithelial cells are embedded within an adipose and
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Book 1988 to date, easily available on a single library shelf or by a single personal subscription. We have approached the problem in the following fashion. First, by divid­ ing the oncology literature into specific subdivisions such as lung cancer, genitourinary cancer, pediatric oncology, etc. Second, by a
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Structure and function of the pS2 gene and estrogen receptor in human breast cancer cells,lar cloning of estrogen-responsive MCF-7 cell gene(s) and 2) the isolation of the human ER cDNA. Of interest is both the function of the gene product of an estrogen-responsive gene and its relationship to cell growth, as well as the mechanism by which estrogen and its receptor control the expression
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