Titlebook: Brain Ischemia; Eugene Gusev,Veronica I. Skvortsova Book 2003 Springer Science+Business Media New York 2003 brain.death.depression.glutama

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Thomas G. Dietterich,Ryszard S. MichalskiAcute focal brain ischemia triggers pathological biochemical reactions that take place in all neural tissue cell pools and cause neuronal dysfunction, astrocytosis, microglial activation, and accompanying changes in neutrophils, macrophages, and endothelial cells.

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An Overview of Machine LearningWhen glutamate is released from presynaptic terminals, it diffuses to the postsynaptic terminals where it binds to glutamate receptors, allowing Na. influx that depolarizes the membrane. This opens Ca. channels allowing Ca. to enter the postsynaptic cell. Under ischemic conditions, this process becomes excessive—the “glutamate-calcium cascade”.

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Fuwei Li,Lifeng Lai,Shuguang CuiWhen effective blood flow is recovered before irreversible necrotic damage forms, a stereotype reparative process will have been started in the brain tissue.

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Studies in Computational IntelligenceModern scientific and technological advances give reason to revise the previous pessimistic opinion about the therapeutic possibilities for acute focal brain ischemia, i.e. ischemic stroke.

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Preamble

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Cellular Reactions in Response to Acute Focal Brain IschemiaAcute focal brain ischemia triggers pathological biochemical reactions that take place in all neural tissue cell pools and cause neuronal dysfunction, astrocytosis, microglial activation, and accompanying changes in neutrophils, macrophages, and endothelial cells.