Titlebook: Brain Ischemia; Eugene Gusev,Veronica I. Skvortsova Book 2003 Springer Science+Business Media New York 2003 brain.death.depression.glutama

Mosquito

书目名称Brain Ischemia影响因子(影响力)




书目名称Brain Ischemia影响因子(影响力)学科排名




书目名称Brain Ischemia网络公开度




书目名称Brain Ischemia网络公开度学科排名




书目名称Brain Ischemia被引频次




书目名称Brain Ischemia被引频次学科排名




书目名称Brain Ischemia年度引用




书目名称Brain Ischemia年度引用学科排名




书目名称Brain Ischemia读者反馈




书目名称Brain Ischemia读者反馈学科排名

cathartic

Energy Failure Induced by Brain Ischemia a substrate for cytochrome oxidase, the terminal enzyme of the respiratory chain. The resulting state of hypoxia is the consequence of a complicated multi-stage process involving sequential changes in the properties of the mitochondrial enzyme complexes [.].

Nebulous

Metabolic Acidosis and Ischemic Damagey activation of anaerobic glycolysis and to increased production of lactate and H. that causes the development of lactic acidosis. Modest increase in H. concentration in early stages of ischemia plays a compensatory and adaptive role as it promotes the improvement of perfusion in the penumbral area

Fecal-Impaction

Thrombolysis

Autoimmune Mechanisms of Trophic Dysfunction and Ischemic Brain Damagethey interact via mutual regulation involving neuromediators, neuropeptides, cytokines, trophic factors, and hormones and the corresponding receptor systems [.]. Mutual regulation of the nervous, endocrine, and immune systems determines the reliability of their common functioning. However, this enta

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Reaction of the Stress-Mediating Endocrine System in Response to Acute Brain Ischemiastem caused by the expression of immediate-early genes—by the synthesis of heat-shock (or stress) proteins (HSP) and cytokines, which are immune mediators [., ., ., .] (see Chapters 7 and 8; Fig. 8.1). The stress-mediating response provides the metabolie basis for compensatory and adaptive changes t

尾随

Condyle

Reparative Therapyin tissue. Thus, from the first hours after stroke onset it is necessary to proceed with treatment aimed at the improvement of reparative and regenerative processes, plasticity of brain tissue, and the formation of new associative pathways, along with neuroprotection aimed at prevention of further b

忙碌

Vimal K. Shrivastava,Monoj K. Pradhanant to mention not only its therapeutic, but also preventive value. Correction of delayed consequences of ischemia can lead to retardation of vascular encephalopathy development in the post-stroke period.

Hyperopia

Secondary Neuroprotectionant to mention not only its therapeutic, but also preventive value. Correction of delayed consequences of ischemia can lead to retardation of vascular encephalopathy development in the post-stroke period.