Titlebook: Brain Edema X; Proceedings of the T Hector E. James,Lawrence F. Marshall,Zbigniew Czer Conference proceedings 1997 Springer-Verlag/Wien 199

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MEMS Lorentz Force Magnetometersed from rat cerebral cortices by a two step enzymatic treatment. The monolayers of BCECs were subjected to anoxia for 20 minutes followed by reoxygenation for 3 hours. Cell damage was assessed by measuring the leakage of intracellular lactic dehydrogenase (LDH). The control group was anoxia/reoxygen

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MEMS Lorentz Force Magnetometersate the effects of mannitol in comatose patients on PtiO., PtiCO. and brain tissue pH using Clark-type electrodes implanted into cerebral white matter. In the neurosurgical intensive care unit PtiO., PtiCO., brain tissue pH, arterial blood pressure, intracranial pressure (ICP), cerebral perfusion pr

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Ronald G. Polcawich,Jeffrey S. Pulskampof recent stroke studies. Two magnetic resonance imaging techniques; diffusion-weighted (DW) and magnetization transfer contrast (MTC) imaging were introduced in experimental studies and were shown to have sensitivity for different stages of brain infarct...We used a reproducible middle cerebral art

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Shawn J. Cunningham,Mario Kupnikma cells have revealed that viable astrocytes swell, spatially buffering various metabolites which are increased by the metabolic turmoil following ischemic insults. In the present study, we have studied the temporal profile of ultrastructural changes of astrocytes in the cerebral cortex associated

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What Is the Product? Requirements Analysiscal ischemia. Two hours of tandem occlusion of the middle cerebral artery (MCA) and the conunon carotid artery produced a scattered reduction of SDH activity and corresponding brain edema in the cortex. Recirculation ameliorated the SDH reduction and the scattered pattern disappeared, although the b

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Design for Back-end-of-Line Processes cerebral ischemia (GCI) by intravital fluorescence microscopy. Four days after 15 min of bilateral common carotid artery occlusion the ischemic tissue damage was histologically analysed in selectively vulnerable areas of the brain. The frequency of Rhodamine 6G labeled leukocytes rolling along (“ro

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What Is the Product? Requirements Analysiscerebrovascular reserve capacity after the brain ischemia. The experiments were carried out in rabbits. Administration of acetazolamide (Diamox) of 20 mg/kg was performed in all experimental animals and the blood flow velocity in the middle cerebral artery (MCA) and the basilar artery (BA) was measu

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