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Titlebook: Brain Edema X; Proceedings of the T Hector E. James,Lawrence F. Marshall,Zbigniew Czer Conference proceedings 1997 Springer-Verlag/Wien 199

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期刊全称Brain Edema X
期刊简称Proceedings of the T
影响因子2023Hector E. James,Lawrence F. Marshall,Zbigniew Czer
视频video
发行地址comprehensive survey of the present state of the art * directions for further research.
学科分类Acta Neurochirurgica Supplement
图书封面Titlebook: Brain Edema X; Proceedings of the T Hector E. James,Lawrence F. Marshall,Zbigniew Czer Conference proceedings 1997 Springer-Verlag/Wien 199
影响因子This volume is a compilation of papers presented at the Tenth International Symposium on Brain Edema held on October 20-23, 1996, in San Diego, California. This follows the sequence of meetings that was initiated 31 years ago in the First International Symposium held in Vienna. Subsequent symposiums were held in Mainz, Montreal, Berlin, Groningen, Tokyo, Baltimore, Bern, and Tokyo CY okohama). A considerable number of papers was chosen from over 100 papers that were received. The organizers wish to thank the Advisory Committee for the excellent work done in selection of the papers. We also wish to thank all the persons who contributed to the success of the Tenth International Symposium, especially the staff who worked behind the scenes. These papers were reviewed, edited, approved or disapproved by the Editorial Board. Those manuscripts that were felt not pertinent to this publication were not accepted by the Editorial Board. Therefore, the excellent quality of those that are in the book are a reflection of the authors‘ dedication and work and that of those of the Editorial Board in their review process. For the reader‘s convenience, the papers are structured according to the vario
Pindex Conference proceedings 1997
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https://doi.org/10.1007/3-540-32382-1hown to cause disruption of blood-brain barrier (BBB) by destruction of capillary endothelial cell membrane. However, the exact mechanism of BBB breakdown by cerebral ischemia/reperfusion remains undetermined..The aim of the present study is to clarify the mechanism of intracellular calcium ion ([Ca
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Effects on Measurement Accuracy,leading to neuronal injury. The effects of hyperglycemia on another possible primary target, the cerebral microvasculature, is examined in this study. Focal cerebral ischemia was achieved by thread occlusion of the middle cerebral artery (MCA). Preischemic hyperglycemia was induced by intraperitonea
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https://doi.org/10.1007/978-1-4419-6020-7brain edema formation. Preischemic hyperglycemia was induced by intraperitoneal administration of D-glucose solution. Laser-Doppler flowmetry, indicated that after the initial decline in blood flow with carotid occlusion (36 ± 4% of preischemic), hyperglycemic but not control rats showed a further p
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