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Titlebook: Brain Chemistry and Mental Disease; Proceedings of a Sym Beng T. Ho,William M. McIsaac Conference proceedings 1971 Springer Science+Busines

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https://doi.org/10.1007/978-3-663-00234-5966; Eberle and Eiduson 1968). More specifically, we believe as others do that a knowledge of how the biosynthesis, tissue levels, and catabolism of these amines are regulated during brain development may lead to a greater understanding of the biochemical and behavioral individuality exhibited by hi
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https://doi.org/10.1007/978-88-470-1905-8t they may be involved in the development of certain affective disorders. Most of the agents used in the treatment for depression and mania are known to affect either the synthesis, storage, and release of catecholamines or the central adrenergic receptors. These brief considerations are sufficient
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L‘adolescent atteint de cancer et les siension whether low brain serotonin (5-HT) or norepinephrine (NE) is specifically responsible for this abnormal state. The evidence that elevating the level of brain 5-HT alleviates depression was first presented by Coppen, Shaw, and Farrell (1963). They reported that administration of tryptophan to dep
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https://doi.org/10.1007/978-2-287-98886-8 concentrations of acetylcholine (MacIntosh 1941) and the enzymes required for its synthesis (Feldberg and Vogt 1948; Hebb and Silver 1956) and catabolism (Burgen and Chipman 1951) are found in this structure. The unusually large concentration of dopamine in the caudate and other basal ganglia promp
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https://doi.org/10.1007/978-2-287-98886-8rosine (α-MPT) in the affective disorders and on the biochemical pharmacology of these agents in man and in the laboratory animal. These studies were prompted by the catecholamine hypothesis, which remains of central importance in current research in the affective disorders (Bunney and Davis 1965; S
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