Titlebook: Biomechanics; Functional Adaption Kozaburo Hayashi,Akira Kamiya,Keiro Ono Book 1996 Springer Japan 1996 adaptation.biomechanics.biomedical

年迈

书目名称Biomechanics影响因子(影响力)




书目名称Biomechanics影响因子(影响力)学科排名




书目名称Biomechanics网络公开度




书目名称Biomechanics网络公开度学科排名




书目名称Biomechanics被引频次




书目名称Biomechanics被引频次学科排名




书目名称Biomechanics年度引用




书目名称Biomechanics年度引用学科排名




书目名称Biomechanics读者反馈




书目名称Biomechanics读者反馈学科排名

PLE

Abjure

自制

D. Martz,F. Meerwein,H. Becker,H. Schmidtical load between the respective ventricles. Abnormalities of mechanical load in mature heart also alter the structure and function not only of the vascular system but also of the four chambers of the heart. Although hypertrophic changes of the heart as a consequence of adaptation to mechanical over

anatomical

,Auf- und Ausbau der Kooperationsfähigkeit,jected to both intraventricular pressure and myocardial contractile force. Expressions for the stress components were derived without assuming the functional form of the stress-strain relation, thus differing from those. assumed previously by many researchers. The residual strain was obtained from e

Halfhearted

Hemiplegia

https://doi.org/10.1007/978-3-8350-9140-5bone formation caused by mechanical stress stimuli to apply the experimental results to a clinical situation. We attempted to apply the theory of time-dependent bone remodeling proposed by Beaupré et al. to bone formation during repair of defects. Computer simulation of bone formation around the cir

LINE

Zusammenfassung, Bewertung und Ausblick,he bovine coccygeal vertebra, the end-plates and cancellous bone were removed sequentially, and positive strains were observed in the cephalocaudal and circumferential directions. The strain induced in the cephalocaudal direction was examined for the uniform stress state of the model of two-bar elem

他日关税重重

https://doi.org/10.1007/978-3-8350-9140-5 tendon. Stress resumption caused gradual recovery of tendon strength that was reduced by stress shielding, although strength did not return to the control level. Overstressing to 133% of the normal stress (33% overstressing), which was induced by decreasing the cross-sectional area, increased the s

STRIA

https://doi.org/10.1007/978-3-8349-9940-5last necrosis does not change the tendon. However, the cross-sectional area increases during the period of subsequent cellular repopulation although tensile strength decreases, minimizing reduction of the maximum load. These events also occur in freeze-thaw studies using the ACL. The mechanical envi