Titlebook: Biomarkers in Cardiovascular Disease; Vinood B. Patel,Victor R. Preedy Reference work 2016 Springer Science+Business Media Dordrecht 2016

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Stefan Schanzenbächer Dr. phil. provide long-term cardiovascular organ protection. In recent years, several trials have shown that drugs with promising effects on the on-target risk factor failed to improve long-term cardiovascular protection. One explanation for these failures is that a drug does not only affect the risk factor

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Ischemic-Stroke

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https://doi.org/10.1007/978-3-322-97602-4ase, heart disease associated with pressure overload, heart disease associated with volume overload, and intrinsic myocardial disease or cardiomyopathy. Collagen, types I and III, is the principal structural protein found in the myocardium, and its pro- or telopeptides are released into the circulat

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Michael Stiels-Glenn,Penelope Glennve to measure its concentration in blood. Circulating PCSK9 exists in several forms – its parent form and a furin-cleaved form and a low-density lipoprotein (LDL)-bound form and a free form. The furin-cleaved and LDL-bound forms are much less active. Most studies have not clearly distinguished among

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https://doi.org/10.1007/978-3-662-12178-8ent functions in human physiology and pathophysiology. Most studies suggest a biphasic effect of fetuin-A depending on the stages of atherosclerosis. Serum levels of fetuin-A are decreased in cases of acute inflammation. Therefore, it is known as a negative acute-phase protein. Fetuin-A inhibits ins