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Titlebook: Biomarkers for Preclinical Alzheimer’s Disease; Robert Perneczky Book 2018 Springer Science+Business Media, LLC 2018 Epidemiology.imaging.

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https://doi.org/10.1007/978-3-322-94349-1Alzheimer’s disease (pAD). We analyze putative models of pAD, covering both genetic and clinical aspects of pAD. Studies on cognitively healthy individuals carrying the APOE-ε4 allele and patients with mild cognitive impairment have been included. To address methodologies, studies on metabolism and
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https://doi.org/10.1007/978-3-322-94349-1ddition to a description of amyloid PET itself, topics such as early and differential diagnosis of AD are discussed. Measuring amyloid in CSF as an alternative method to amyloid PET and the associations between cerebral amyloid load and the apolipoprotein E genotype are also covered in this section.
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Konflikthafte Vergemeinschaftungse (AD), Parkinson’s disease without (PD) and with later dementia (PDD), Lewy body dementia (LBD), frontotemporal dementia (FTD), and corticobasal degeneration (CBD). Aggregated tau tangles, the result of hyperphosphorylation, is a pathological characteristic of a group of neurodegenerative conditio
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Book 2018e (AD). The chapters in this book are separated into five parts. Part one explores the reasons why we need improved approaches for early detection and diagnosis of AD. Part two describes clinical and research techniques for preclinical AD diagnosis. Part three looks at the current methods in use to
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