Titlebook: Biology of the Mammary Gland; Jan A. Mol,Roger A. Clegg Book 2002 The Editor(s) (if applicable) and The Author(s), under exclusive license

情感

Paracrine Mechanisms of Mouse Mammary Ductal Growth,h targeted disruption of ERa or ERb has emphasized the importance of ERa in mammary gland development. In the mouse mammary gland, ERα are expressed in both epithelial and stromal cells (Kurita and Cunha, unpublished), which raises the possibility that the growth and morphogenetic effects of estroge

Gingivitis

corporate

The PEA3 Group of ETS-related Transcription Factors, the ETS-domain, which binds to sites containing a central 5,-GGAA/T-3, motif. They can be sub-classified primarily because of the high amino acid conservation in their ETS-domains and, in addition, in the conservation of other domains generally characterized as transactivating. This is the case for

COST

CHYME

The Elf Group of Ets-Related Transcription Factors,y-specific. It seems that a complex interplay of transcription factors may be responsible for mammary expression. Our challenge is to determine whether ELF3 anhion of mammary-specific genes ., and whether they cooperate with other stimuli, such as prolactin. Because the mammary gland is not a static

感染

The Role of Stat3 in Apoptosis and Mammary Gland Involution,cytokines and growth factors. This family of signalling molecules have been implicated in growth, differentiation, survival and apoptosis. In this article, we will review work which highlights the role of individual STAT factors in mammary gland and demonstrate the value of genetically modified mice

forager

Synergistic and Antagonistic Interactions of Transcription Factors in the Regulation of Milk Proteites for distinct classes of transcription factors, which either co-operate or are antagonistic. In addition, the activity of some of these factors is individually control-led by diverse extracellular signals. A well studied paradigm for a synergistic co-operation is the activation of beta-casein gen

不妥协

Modicum

Chromatin Remodeling in Hormone-Dependent and - Independent Breast Cancer Cell Lines,ression. An increasing number of evidence links defects in chromatin remodelling machinery and cancer. Our aim is to elucidate the role of chromatin structure in the control of the expression of hormone-induced genes in breast cell lines oestrogen-dependent or -independent for growth. Mammary tumour

放逐某人